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Structure of transition classes for factor codes on shifts of finite type
ALLAHBAKHSHI, MAHSA,HONG, SOONJO,JUNG, UIJIN Cambridge University Press 2015 Ergodic theory and dynamical systems Vol.35 No.8
<P>Given a factor code ${\it\pi}$ from a shift of finite type $X$ onto a sofic shift $Y$, the class degree of ${\it\pi}$ is defined to be the minimal number of transition classes over the points of $Y$. In this paper, we investigate the structure of transition classes and present several dynamical properties analogous to the properties of fibers of finite-to-one factor codes. As a corollary, we show that for an irreducible factor triple, there cannot be a transition between two distinct transition classes over a right transitive point, answering a question raised by Quas.</P>
Allahbakhshi Maryam,Mosaferi Mohammad,Mahmoodi Niyaz Mohammad,Kazemian Hossein,Aslani Hassan 한국화학공학회 2023 Korean Journal of Chemical Engineering Vol.40 No.12
MIL-53(Fe) as a three-dimensional MIL was prepared with different precursors-to-solvent ratios and denoted as MIL53B and MIL53C. All as-synthesized samples were modified using 3-amino propyl trimethoxy silane (organosilane) for synthesizing MIL53B/NH2(0.10), MIL53B/NH2(0.20), MIL53B/NH2(0.30), MIL53C/NH2(0.10), MIL53C/NH2(0.20), and MIL53C/NH2(0.30), respectively. The XRD, SEM, EDS, and FTIR techniques were used to characterize materials. The adsorption ability of the materials for the adsorption of dye (Direct Red 23) was studied. The effect of operational parameters was evaluated. The kinetics and isotherm of pollutant removal obeyed pseudo-second-order kinetic and the Langmuir models, respectively. The row and modified MIL53B indicated the adsorption capacity of 1,375 and 4,989 mg/g, respectively. The high pollutant removal ability and fast adsorption rate ascertain that the modified MIL53 could be considered as a dye adsorbent and potentially other organic molecules with similar structures from contaminated water.
Edentulous child with Allgrove syndrome: a rare case report
Vahedi, Mohammad,Fathi, Shima,Allahbakhshi, Hanif The Korean Pediatric Society 2016 Clinical and Experimental Pediatrics (CEP) Vol.59 No.11
Triple-A syndrome, also known as Allgrove syndrome, is a rare autosomal recessive disorder. The 3 features of this syndrome are achalasia, adrenal insufficiency, and alacrima. Achalasia could be the first manifestation of the triple-A syndrome; however, its etiology is unclear. Alacrima is generally asymptomatic but can be detected by obtaining patient history. Although adrenal insufficiency could have manifestations such as asthenia, it might be wrongly diagnosed as muscle fatigue. Vitamin D and calcium supplements are usually prescribed for the prevention of osteoporosis. Neurologic manifestations could be present in adults. In some individuals with this disorder, genetic examination indicates mutations in both alleles of the AAAS gene, which encodes a special 546-amino-acid protein designated ALADIN, and in chromosome 12q13. The genetic cause of the triple A syndrome in some patients who do not have an identified mutation is unknown. While very few such cases have been reported till date, one such case was presented to us as an edentulous child.
Edentulous child with Allgrove syndrome: a rare case report
Mohammad Vahedi,Shima Fathi,Hanif Allahbakhshi 대한소아청소년과학회 2016 Clinical and Experimental Pediatrics (CEP) Vol.59 No.11
Triple-A syndrome, also known as Allgrove syndrome, is a rare autosomal recessive disorder. The 3 features of this syndrome are achalasia, adrenal insufficiency, and alacrima. Achalasia could be the first manifestation of the triple-A syndrome; however, its etiology is unclear. Alacrima is generally asymptomatic but can be detected by obtaining patient history. Although adrenal insufficiency could have manifestations such as asthenia, it might be wrongly diagnosed as muscle fatigue. Vitamin D and calcium supplements are usually prescribed for the prevention of osteoporosis. Neurologic manifestations could be present in adults. In some individuals with this disorder, genetic examination indicates mutations in both alleles of the AAAS gene, which encodes a special 546-amino-acid protein designated ALADIN, and in chromosome 12q13. The genetic cause of the triple A syndrome in some patients who do not have an identified mutation is unknown. While very few such cases have been reported till date, one such case was presented to us as an edentulous child.