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Russell Body Lesions of the Colon: Case Report and Literature Review
( W. Keith Tan ),( Rachel Smith ),( Anthony George ),( Anita Gibbons ),( Syed Shaukat ),( Rizwan Kassam ),( Phil Roberts ) 대한소화기학회 2021 대한소화기학회지 Vol.77 No.6
Russell bodies (RB) are rare manifestations within the lower gastrointestinal tract. To date, there are only three other reported cases of RB lesions of the colon; two were polyps, and the third was a case of a multifocal RB lesion of the gastrointestinal tract. This paper reports a case of a tubulovillous adenoma with RB of the sigmoid colon in a patient diagnosed incidentally as part of the UK National Health Service Bowel Cancer Screening Programme. A thorough hematological investigation is required to exclude hematological malignancies because of its association with plasma cell neoplasm. These lesions should undergo clonality analysis to exclude the monoclonal proliferation of plasma cells. Ideally, a bone marrow aspirate and investigations for amyloidosis should be performed to exclude underlying hematological malignancies. (Korean J Gastroenterol 2021;77:309-312)
Patterns of Firm Entry and Exit in Texas Home Care Industry
Dakshina G. De Silva,정호진,Robert McComb,Anita R. Schiller 한국산업경제학회 2021 한국산업경제학회 정기학술발표대회 논문집 Vol.2021 No.8
퇴원 또는 지리적 변동의 결정 요인에 대한 가정건강관리 서비스의 영향에 관심을 가졌던 기존 연구와 달리, 본 연구에서는 가정건강관리 서비스 공급자의 진입 및 퇴출 유형을 조사하였다. 특히 미국 텍사스 주의 상세한 데이터를 사용하여 가정방문간호 서비스 제공자의 진입, 성장 및 퇴출 유형의 결정 요인을 분석하였다. 실증분석을 통해 시장의 진입하는 방문간호서비스 공급자는 보완적인 의료 서비스를 제공하는 다른 보건시설들로 인해 국지화경제 효과를 누릴 수 있음을 발견하였다.
D'Souza, Rena N.,Andriana Cavender,Rajita Sood,Roy Tarnuzzer,Dickinson, Douglas P.,Anita Roberts,John Letterio Korean Academy of Oral Biology and the UCLA Dental 1998 International Journal of Oral Biology Vol.23 No.3
To better understand the precise physiologic functions of TGF- β1 in odontogenesis, we have performed a detailed analysis of developing and adult dentition in mice with a targeted deletion of the TGF-β 1 gene. TGF-β1(-/-) mice derived from TGF- β1(+/-) matings receive maternally transferred TGF-β1, but die by three weeks of age from multifocal inflammation (Shull et al., 1992; Kulkarni et al., 1993). In these mice and neonates from a TGF-β1(-/-) female that did not receive maternal TGF-β1, the earlier stages of odontogenesis appeared unaltered. Furthermore, the absence of TGF-β1 did not affect the patterns and levels of expression of related isoforms TGF-β2 and TGF-β3, as shown by in-situ hybridization and competitive RT-PCR analyses. However, histopathologic analysis of adult dentition in TGF-β1(-/-)mice, whose survival was prolonged with dexamethasone, showed extensive pathlolgy in dental and paradental tissues. In addition to inflammation, we observed a marked attrition of tooth matrices in TGF-β1(-/-) molars when compared to TGF-β1(+/+) and TGF-β1(+/-) littermates. To determine whether the phenotypic changes in teeth are primarily caused by the deficiency in TGF-β1 rather than the secondary effects of inflammation, we studied dentition in TGF-β1(-/-) mice backcrossed onto immunodeficient backgrounds using histochemical, radiographic and biomechanical methods. These analyses revealed significant differences in mineral content and hardness quality in TGF-β1(-/-) molars as compared to their control littermates, implying that the impairment in formation of dental mineralized tissues is linked directly to the absence of TGF-β1. Our findings hence demonstrate the importance of TGF-β1 in cellular functions at advanced stages of odontogenesis, in particular, the biomineralization of tooth matrices. In addition these studies indicate a non-redundant, isoform-specific role of TGF-β1 in the immune homeostasis of oral tissues.