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민병우,고준석,백승기,반종석 대한마취과학회 1982 Korean Journal of Anesthesiology Vol.15 No.4
Malignant hyperthermia still carries a high mortality despite of the increased understanding of the problem involved. A 33 year old relatively healthy male patient was admitted to this hospital via emergency room because of panperitonitis due to peptic ulcer perforation. We experienced acase of malignant hyperthermia which developed 20 minutes after induction of general anesthesia with thiopental sodium, succinyl choline chlovide, halohane, N_2O and O_2. The body temperature (esophageal) rose altupthy up to 41.2℃ and continued as a high fever for about 3 hours. The blood pressure and heart rate also increased and ventricular dysrrhythmia appeared. The etiology, triggering factors, clinical features, diagnosis treatment and rafe anesthesia of malignant hyperthermia are discussed.
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Pentoxifylline 및 Adenosine이 E. coli 내독소에 의한 폐상해에 미치는 영향
민병우,곽정식,손태중 慶北大學校 醫科大學 1992 慶北醫大誌 Vol.33 No.1
This study was carried out to investigate the effects of pentoxifylline and adenosine on the pulmonary injury induced by E. coli endotoxin. The counts of circulating neutrophils in peripheral blood and pulmonary sequestration of neutrophils after injection of endotoxin, endotoxin with pentoxifylline, or endotoxin with adenosine, additionally electron microscopy of the lungs were carried out. To account the circulation neutrophils, six white rabbits were intravenously injected 4㎎/㎏ of E. coli 026 : B6 lipopolysaccharide, and then counted circulating neutrophils at 0, 1, 2, 4, and 8 hours after injections. Another six were injected 4㎎/㎏ of endotoxin simultaneously with 100㎎/㎏ of pentoxifylline (40㎎/㎏ at 0 hour and 20㎎/㎏ at 1, 2, and 4 hours after first injection), then neutrophils were counted at 0, 1, 2, 4, and 8 hours after injection of the endotoxin. The third six were injected 4㎎/㎏ of endotoxin simultaneously with 50㎎/㎏ of adenosine(20㎎/㎏ at 0 hour and 10㎎/㎏ at 1, 2, and 4 hours after first injection), also the neutrophils were counted at 1, 2, 4, and 8 hours after endotoxin injection. Neutrophilic pulmonary sequestration was counted at 8 hours after injection of endotoxin, endotoxin with pentoxifylline, or endotoxin with adenosine. Electron microscopy of the lungs was carried out at 1, 4, and 8 hours after injections in each group. The results were as follows: The number of circulating neutrophils was markedly decreased at 1 hour after endotoxin injection and sustained until 8 hours after injection. In the pentoxifylline and adenosine injected groups, circulating neutrophils were decreased in number at 1 hour, however, nearly recovered at 8 hours after injections. Pulmonary sequestration of neutrophils was marked in endotoxin injected group. In contrast, endotoxin with pentoxifylline and endotoxin with adenosine injected groups demonstrated mild sequestration of the neutrophils. Ultrastructural changes of the lungs were swelling, formation of cytoplasmic projections, and seperation of the cytoplasm from the basement membrane in the endothelium, swelling of type Ⅱ pneumocytes, and interstitial edema appeared in pentoxifylline and adenosine injected groups. According to the above results, it is concluded that pentoxifylline and adenosine inhibit pulmonary sequestration of the neutrophils in endotoxemia and suppress pulmonary injury induced by endotoxin.
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閔炳佑,趙誠璟,朴大源,李相華 대한마취과학회 1977 Korean Journal of Anesthesiology Vol.10 No.3
During any anesthesia, the patient's general condition must be observed continuously but even more so in patients with cardiac disease. Cardiac dysphthmias may he observed by continuous ECG monitoring in the operating theatre even in normal patients but the intractable ventricular dysrythmias are serious problems and a cause of great goncern. Any serious cardiac condition must be ascertained and treated at once with various antiarrhythmic agents or other techniques, such as D.C shock, carotid sinus pressure, eye-ball pressure, positive inotropics, or atropine, etc. With an anesthesia for an intractable ventricular dysthythmia, the authors found that this could mot be restored to a normal rhythm by means of various antiarrhythmic agents. Several months previously, the patient had been in another hospital in a state of septic shock which apparently affected the myocardium, resulting in ischemic cardiomyopathy. Immediately after the completion of the operation and anesthesia, the patient, with the aid of specific drugs and chest thumps, regained consciousness with relative normal cardiac rhythm. We would conclude that continuous ECG monitoring, close observation, as well as specific antiarrhythmic agents, are most essential during anesthesia of a patient with cardiac disease.
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α_2-수용체 효현제와 Benzodiazepine류가 병아리 수면작용에 미치는 영향
민병우,손의동,김중영 慶北大學校 醫科大學 1989 慶北醫大誌 Vol.30 No.2
부화된지 이틀된 병아리에 α_2-수용체 효현제를 투여하여 나타나는 수면작용과 benzodiazepine류에 의한 수면작용이 서로 관련이 있는지를 검토하기 위하여 수면기간과 발현시간을 비교하여 다음과 같은 결과를 얻었다. α_2-수용체 효현제인 guanabenz(0.3, 1㎎/㎏) 근육주사로 나타나는 수면발현시간은 용량증가에 따라 짧아지고 xylazine(3,10㎎/㎏)은 변화가 없었으나, 수면기간은 모두 용량증가에 따라 길어다. 대조적으로 phebylephrine 투여시는 고용량에서도 수면작용이 나타나지 아니하였다. Benzodiazepine류인 diazepam(3,10㎎/㎏), lorazepam(3,10㎎/㎏)은 변화가 없었으나, 수면기간은 모두 용량증가에 따라 길어졌다. 대조적으로 phenylephrine 투여시는 고용량애서도 수면작용이 나타나지 아니하였다. Benzodiazepine류인 diazepam(3,10㎎/㎏), lorazepam(3,10㎎/㎏) 혹은 flurazepam(10,30㎎/㎏) 투여시 용량증가에 따라 수면지속시간은 길어졌으나 발현시간은 일정하지 않았다. Yohimbine 전처치에 의해 guanabenz나 xylazine 투여에 의한 수면작용은 완전히 봉쇄되었으나 diazepam, lorazepam 혹은 flurazepam에 의한 수면작용은 영향을 받지 않았다. 그리고 diazepam 전처치에 의해서 guanabenz나 xylazine 투여에 의한 수면작용은 영향을 받지 않았다. 이와 같은 결과로 보아서 guanabenz나 xylazine으로 유도된 수면작용은 중추의 α_2-수용체와 밀접한 관계가 있으나, diazepam, lorazepam 혹은 flurazepam에 의한 수면작용은 α_2-수용체와는 관계가 없는 것으로 사료된다. Hyponsis produced by alpha-2 agonists, quanabenz and xylazine was compared with the hypnosis induced by benzodiazepines in 2-day-old chicks. Onset time of sleep induced by guanabenz was more shortened by 1 ㎎/㎏ than 0.3 ㎎/㎏, but the onset time by dosage of xylazine had no difference. Duration of sleep by guanabenz and xylazine was more prolonged by high dose than by low dose, but phynylephrine did not produce sleep. Onset time of sleep induced by diazepam(3, 10㎎/㎏) and flurazepam(10, 30㎎/㎏) was more shortened by high dose than low dose. Duration of sleep induced by all three kinds of benzodiazepines was more prolonged by high dose than low dose. Sleep induced by guanabenz and xylazine but not benzodiazepines inhibited by yohimbine pretreatment, and it was not affected by diazepam pretreatment. These results show that sleep by guanabenz and xylazine produce by alpha-2 receptor stimulation and the action do not affect to benzodiazepine-induced sleep.
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