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김효수,김창원,Yejin Kim,Minsoo Kim,Wenhua Piao,Jeasung Gee 한국화학공학회 2014 Korean Journal of Chemical Engineering Vol.31 No.4
This paper proposes real-time control strategies that can be applied in a full-scale advanced phase isolationditch (APID) process. Real-time operation mode control (OMC) and aeration section control (ASC) strategies weredeveloped to cope more stably with fluctuations in the influent loading and to increase the nitrification and denitrificationreactions within the entire volume. The real-time OMC and ASC strategies were evaluated using mathematicalmodels. When the NH4-N in the reactor was maintained at a high level, appropriate control actions, such as continuingthe aeration state, stopping the influent inflow and increasing the aeration section, were applied in the APID process. In contrast, when the NOX-N in the reactor was maintained at a high level, the non-aeration state, influent inflow, anddecreased aeration section were continued. It was concluded that stable operation in the APID process could be achievedby applying real-time OMC and ASC strategies developed in this study.
김효수,김일환,Kim, Hyo-Soo,Kim, Il-Hwan 사상체질의학회 2004 사상체질의학회지 Vol.16 No.3
1. Objectives Facial Palsy(Bell's palsy) is a common disease in oriental medicine. In Sasang Constitutional Medicine, Taeumin have a basic condition by Ganyeol as their inherent symptomatic phamacology. This case is a study about Taeumin's facial palsy in Ganyeol conditions. 2. Methods In treatment of facial palsy, especially in acute stage, We prescribe Yeoldahanso-tang(熱多寒少湯) when a patient with facial palsy is diagnosed as Taeumin by their own characters. 3. Conclusions This case-study shows an efficient results by using Yeoldahanso-tang(熱多寒少湯) in treatment of facial palsy compared with other known treatments.
활성슬러지 모델 수정을 통한 동시 질산화·탈질 반응 해석
김효수(Hyo Su Kim),김예진(Ye Jin Kim),이성학(Sung Hak Lee),문태섭(Tae Sup Moon),최재훈(Jae Hoon Choi),김창원(Chang Won Kim) 大韓環境工學會 2008 대한환경공학회지 Vol.30 No.2
동시 질산화·탈질은 낮은 DO 농도로 유지되는 동일한 반응조에서 질산화 반응과 탈질 반응이 동시에 발생함을 의미한다. 동시 질산화 탈질 반응을 모사할 수 있는 몇몇 수학적 모델들이 개발되었지만, 모델 구조가 복잡하거나 모델을 적용하기 위한 다양한 제반 지식을 얻어야만 정확한 결과를 얻을 수 있어 범용적인 모델 적용에 한계점이 있는 단점이 있었다. 이와 같은 문제를 해결하기 위해, 동시 질산화·탈질 반응이 반응기 내 DO 농도의 부분적 부재로 발생한다는 가정 하에, 만약 활성슬러지 모델을 사용하여 동시 질산화·탈질 반응의 거동을 해석할 수 있다면, 모델의 구조가 다른 개발된 모델들보다 복잡하지 않고, 다양한 운전 조건에서 모델이 활용될 수 있을 것으로 판단하였다. 하지만 기존의 활성슬러지 모델로는 호기 조건에서 발생하는 탈질 반응을 표현하기 어려운 점이 있기 때문에, 본 연구에서는 활성슬러지 모델을 수정함으로써 동시 질산화·탈질 반응을 해석하고자 하였다. 활성슬러지 모델 No.1(ASM1)이 선택이 되어 탈질 반응식이 수정되었으며, 수정된 ASM1의 시뮬레이션 결과는 측정값의 거동을 잘 모사하였다. 이를 통해 수정된 ASM1은 실험 결과에 기반하여 구한 hg의 값과 호기 조건에서의 탈질 반응을 모사하기 위해 수정된 Monod 식의 영향으로 모델의 구조가 본 연구의 실험 결과에서 확인된 동시 질산화·탈질 반응을 해석할 수 있도록 구성되었다고 사료된다. Simultaneous nitrification and denitrification means that nitrification and denitrification occur concurrently in the same reaction vessel under low DO concentration. Some mathematical models developed to simulate simultaneous nitrification and denitrification reaction, but they have the complex model structures or have limitations of model application. To solve these problems, if possible that predict the behavior of simultaneous nitrification and denitrification reaction by activated sludge model, structures of the model is less complex than previous models and applies the various operation conditions. But original activated sludge models have difficulties in representing the denitrification reaction under aerobic condition. So the aim of this study is to interpret simultaneous nitrification and denitrification reaction by modifying activated sludge model. Original activated sludge model No.1(ASM1) was selected and modified. The simulation result in modified ASM1 predicted appropriately for the measured data. This indicates the structures of ASM1 are properly improved for interpretation of simultaneous nitrification and denitrification reaction.
김인호(In Ho Kim),박선양(Seon Yang Park),이종태(Jong Tai Lee),방수미(Soo Mee Bang),김효수(Hyo Su Kim),김병국(Byoung Kook Kim),김노경(Noe Kyeong Kim),박성섭(Seong Sub Park),조한익(Han Ik Cho),정해영(Hae Young Jeong),유욱준(Ook Joon Yo 대한내과학회 1996 대한내과학회지 Vol.51 No.6
N/A Objectives: Thromboembolism is a serious medical problem causing considerable morbidity and mortality. Major clinical risk factors for thrombosis included surgery, fracture, malignancy, old age, immobilization and the use of oral contraceptives. In the last several decades, substantial progress has been made in identifying hereditary factors predisposing to thrombosis. The genetic defects known to be associated with thrombophilia are deficiencies of antithrombin 3, protein C, protein S, dysfibrinogenemia and resistance to the anticoagulant action of activated protein C. We have elucidated the characteristics of heriditary thrombophilia of the Korean patients. Methods: The clinical profiles of 48 patients with heriditary thrombophilia (12 cases of our hospital and 36 cases reported previously in Korea) were analyzed. The underlying hemostatic abnormalities about antithrombin 3, Protein C, Protein S, activated protein C, fibriongen were investigated. Family studies of 6 patients of our 12 patients were done. Nucleotide sequences of antithrombin 3 genes of 2 patients were studied. Results: 1) Seven patients (58%) among our 12 patients had thrombotic onset before fifth decades, and 5 patients developed thromboses at their third decades. 2) Pulmonary embolisms were diagnosed in 10 cases (83%) among our 12 cases. Deep vein thromboses of lower extremities were 8 cases (67%) and usually rare site thromboses like a portal vein thrombosis occurred frequently (8 cases, 67%). Arterial thromboses occurred in two cases (17%). Hereditability of 5 families were confirmed by family study. 3) Analysis of 48 cases showed that protein C deficiency (12 cases, 25%) and protein S deficiency (19 cases, 40%) occurred relatively frequently. 4) Neither Resistance to activated protein C nor mutation in the factor V gene was demonstrated in Korean patients with deep vein thrombosis and normal persons. This result suggests that activated protein C resistance may be extremely rare in Korean population. 5) Two new mutations of antithrombin 3 genes were identified in two patients via nucleotide sequencing, and they were named 'AT 3 Seoul' and 'AT 3 Kosung' respectively. Conclusion: We elucidated clinical and laboratory characteristics of hereditary thrombophilia in Korea. Hereditary thrombophilia were not uncommon in Korean patients with deep vein thrombosis, with/without pulmonary embolism. Strong suspicion for hereditary thrombophilia may lead to correct diagnosis and appropriate treatment in these patients.