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퍼지 신경망을 이용한 ATM 망의 호 수락 제어 시스템의 설계
유재택(Yoo Jae Taek),김춘섭(Kim Choon Seop),김용우(Kim Yong Woo),김영한(Kim Young Han),이광형(Lee Kwang Hyung) 한국정보처리학회 1997 정보처리학회논문지 Vol.4 No.8
In this paper, we proposed the FNCAC (fuzzy neural call admission control) scheme of the ATM networks which used the benefits of fuzzy logic controller and the learning abilities of the neural network to solve the call admission control problems. The new call in ATM networks is connected if QoS(quality of service) of the current calls is not affected due to the connection of a new call. The neural network CAC(call admission control) system is predictable system because the neural network is able to learn by the input/output pattern. We applied the fuzzy inference on the learning rate and momentum constant for improving the learning speed of the fuzzy neural network. The excellence of the proposed algorithm was verified using measurement of learning numbers in the traditional neural network method and fuzzy neural network method by simulation. We found that the learning speed of the FNCAC based on the fuzzy learning rules is 5 times faster than that of the CAC method based on the traditional neural network theory.
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유세포분석기로 확진한 남매에서 발생된 혈소판 무력증 2 예
김동진,박경식,김재삼,이숙경,성낙현,백석기,허완재,임채호,현정례,박훈기,남이현,김춘섭 대한내과학회 1996 대한내과학회지 Vol.51 No.2
Glanzmann's thrombasthenia is a rare autosomal recessive genetic disorder of platelet function. It is caused by the deficiency or abnormality of the major platelets glycoprotein(GP)IIb-IIIa complex. Glanzmann's thrombasthenic platelets show no aggregation in response to the stimulation of ADP and platelet agonist(epinephrine, collagen), but it show aggregation in response to ristocetin. The platelet membrane glycoprotein IIb-IIIa complex is involved in cell to cell interaction and cell to matrix fibronectin interaction. Because the binding of fibrinogen allows platelets to aggregate, the binding of platelets on the subendothelium. The absence of defect of this receptor cause the platelet bleeding disorder called Glanzmann's Thrombasthenia. We experienced two cases of Glanzmann's thrombasthenia in 16-year-old female and 15-year-old male. They are from the same family, but they parents and older sister had no history of bleeding tendency. The 16-year-old female showed frequent epistaxis, showed frequent gum bleeding for the past 5 years, and she was prone to bruise since she was 11-year-old. Her 15-year-old brother showed similar symptom. On platelet aggregation test, mother showed normal response, but her two child showed no response to ADP, epinephrine and collagen, but showed normal response to ristocetin. They were diagnosed as Glannnann's Thrombasthenia by the analysis of Glycoprotein on platelet membrane by Flow-cytometry. The review of the literature was made briefly.
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