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폐렴구균 중증지역사회획득폐렴의 빈도 및 임상적 특징: 비폐렴구균과의 비교
김용균 ( Yong Kyun Kim ),송수희 ( Su Hee Song ),김동규 ( Dong Kyu Kim ),김율빈 ( Rul Bin Kim ),박성훈 ( Sunghoon Park ),정기석 ( Ki Sung Jung ),이석원 ( Seok Won Lee ),박지영 ( Ji Young Park ),안영환 ( Young-hwan An ),장길수 ( Gil 대한내과학회 2012 대한내과학회지 Vol.82 No.1
Limited data are available on severe community-acquired pneumonia (severe CAP or SCAP) caused by Streptococcus pneumoniae in Korea. All patients who were admitted to a tertiary hospital for CAP from January 2007 to December 2008 were reviewed retrospectively, and SCAP was defined by 2007 Infectious Disease Society of America/American Thoracic Society criteria. In total, 94 patients were diagnosed with SCAP (mean age, 73.5 ± 14.3 years; male, 70). Among them, pneumococcal SCAP (P-SCAP) accounted for 24.5%, and non-P-SCAP accounted for 18.1% (four with Pseudomonas aeruginosa, [4.3%]; four with Staphylococcus aureus, [4.3%]), and no organisms were identified in 57.4% of the patients. A history of neoplasm was Less frequent, and the incidence of shock and pneumonia severity index (PSI) scores were Lower in patients with P-SCAP than in those with non-P-SCAP or with SCAP with no organism identified (p = 0.012, 0.023 and 0.007, respectively). Patients with P-SCAP had a Lower rate of treatment failure (p = 0.048) and tended to have Lower in-hospital and 30-day mortalities compared with those with non-P-SCAP. In a multivariate analysis, the history of neoplasm was the strongest independent factor for predicting 30-day mortality (odds ratio, 9.068; 95% confidence interval, 1.856-44.309). SCAP accounted for 24.5% of SCAP cases. P-SCAP was associated with Lower disease severity and a tendency toward better hospital outcomes compared with non-P-SCAP. (Korean J Med 2012;82:52-59)
만성 B형 간염 보균자에서 간염의 급성 악화와 동반된 다발성 관절염
강동훈 ( Dong Hoon Kang ),서영일 ( Young Il Seo ),김율빈 ( Rul Bin Kim ),김호중 ( Ho Joong Kim ),임수영 ( Soo Young Lim ),한지숙 ( Ji Suk Han ),송수희 ( Su Hee Song ),임수경 ( Soo Kyung Lim ),김현아 ( Hyun Ah Kim ) 대한류마티스학회 2013 대한류마티스학회지 Vol.20 No.5
Hepatitis viruses (hepatitis B virus (HBV) and hepatitis C virus) have been associated with development of inflammatory arthritis. Approximately 400 million people worldwide have chronic HBV infection. HBV infection is the one of the most common causes of liver disease, and the prevalence of HBV infection in Korea is almost 6%. Arthritis in patients with HBV can be encountered in two settings: as a rheumatoid arthritis (RA)-like, acute, self-limited polyarthritis during the pre-symptomatic phase of acute hepatitis B, or, more rarely, as arthritis occurring in the context of HBV-associated polyarteritis nodosa (PAN). In both cases, the pathogenesis of arthritis is attributed to the deposition of immune complexes containing viral antigens (HBsAg or HBeAg) and their respective antibodies (anti-HBs and anti-HBe) in synovial tissues. Here we report on a case of polyarthritis associated with reactivation of chronic hepatitis B virus infection with a review of the literature.