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      • KCI등재

        정상월경주기부인의 요중 Luteinizing Hormone의 동태에 관한 면역학적 연구

        김용성(YS Kim),신면우(MW Shin) 대한산부인과학회 1973 Obstetrics & Gynecology Science Vol.16 No.1

        정상월경주기부인의 요중 Luteinizing Hormone의 동태에 관한 면역학적 연구 For the immunologic determination of Luteinizing Hormone (L.H.) in urine during normal menstrual cycle, basic experiments and quantitative assay has been performed using hemagglutination inhibition reaction based upon the cross reaction between the H.C.G.-antiserum to L.H.. Following results were obtained. 1. Human Chorionic Gonadotropin(H.C.G.) supplied by Mann Research Laboratories, New York, N.Y. have been used as antigen for the immunization of rabbits and relative high titer of antiserum was obtained through the effects of Freund`s adjuvant and booster injections. 2. Gel diffusion method of Ouchterlony revealed only one clear precipitation line between the H.C.G. and its antiserum, indicating that the used antigen was relatively pure and the antisera contained negligible amount of no-specific antibody. 3. The sensitivity of hemagglutination inhibition reaction in this study was about 100-150 I.U./L.. 4. Early morning samples of urine were collected daily or every other day from 5 menstrual cycle of 4 normally menstruating women and basal body temperatures were recorded daily for the estimation of the date of ovulation. 5. One volume of urine was treated with 4 volumes of acetone and L.H. containing fraction was washed with one volume of absolute alcohol and ether, then dissolved in 1/10 volume of pH 6.4 phosphate buffered saline, thus concentrating the original specimen 10 times. 6. Quantitative determination of L.H. with serial 1.58-fold dilution of the urine concentrates revealed a single preovulatory peak (300-400 I.U./L.) and relative elevated levels were found several days before and after each peak and the lowest levels of L.H. were found before and after each menses.

      • KCI등재

        임신부사망 및 주생기사망에 관한 임상통계적 관찰

        김용성(YS Kim) 대한산부인과학회 1969 Obstetrics & Gynecology Science Vol.12 No.5

        Maternal mortality in Seoul National University Hospital was analyzed for 6 year and 9 months period, 1962-1968. Durig this period there were 5,975 live births and 20 maternal deaths. The maternal mortality rate per 100,000 live birds was 33.5 There were 240 cases of fetal deaths and 157 cases of neonatal deaths among the 6,225 dirds. Average perinatal mortality was 63.8. The leading causes of maternal mortality in S.N.U.H. were toxemia (30%), hemorrhage (30%) and infection (20%). Complications of abortion and ectopic gestation were responsible for the majority of early pregnancy obstetrical deaths. The principal overall cause in neonatal deaths was prematurity and other causes of neonatal deaths were anoxia, malformation, abnormal pulmonary ventilation and birth trauma. Data analyzed in SNUH show a significant association between prenatal care and maternal and perinatal mortality. The effect of maternal age, parity, gestation, method of delivery and place of delivery were reviewed in association with maternal mortality. The relationship between perinatal mortality and duration of gestation, birth weight, method of delivery, and the time of neonatal death were also reviewed.

      • KCI등재

        만삭자궁 파열 3례

        김기종(KC Kim),이경희(KH Lee),김용성(YS Kim),이진용(JY Lee) 대한산부인과학회 1971 Obstetrics & Gynecology Science Vol.14 No.12

        만삭자궁 파열 3례를 보고하는 바이다. Rupture of the uterus constitues one of the most serious complication of the pregnancy. It is not frequent disease, but it caused 5% of all maternal death. Although the overall incidence has remained fairly constant recently, there has been a realtive change in the number of ruptures attributed to the particular cause. Rupture of the uterus due to obstetrical trauma has shown a marked decrease; this is the result of careful management. The resultant increase in the incidence of cesarean section, however, has brought about a relative increase in incomplete (silent dehiscence) uterine ruptures. We experienced 3 cases of uterine rupture recently: 1-st case was traumatic rupture due to careless use of exytocin by midwife. 2-nd case was spontaneous rupture of cesarean scar during labor. 3-rd case was traumatic rupture of previous cesarean scar by the administration of oxytocin at local clinic.

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