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Growth Characterization of Carbon Nanotubes by Using Optical Interference Oscillations Phenomena
Do-HyungKim,장훈식,Chang-DukKim,Dong-SooCho,Hee-DongKang,Sang-YunLee,Hyeong-RagLee 한국물리학회 2002 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.41 No.5
Various characteristics of the growth of carbon nanotubes (CNTs) were examined by using in-situ optical interference patterns. A dc plasma chemical vapor deposition technique was used to grow the CNTs. Significant interference oscillations were seen during the growth of CNTs. The light absorption in CNTs exponentially decayed with increasing CNT length. From the optical interference curves, the growth rate and the termination length of the CNTs were precisely estimated. The optical interference curves provided considerable in-situ information, such as the length, the alignment, and the related growth properties of the CNTs.
( Yune Jung Park ),( Seong Hye Hwang ),( Seung Hyun Jung ),( Sa Seong Lee ),( Susan Na Choi ),( Seung Ah Yoo ),( Ji Hwan Park ),( Dae Hee Hwang ),( Seung Cheol Shim ),( Chul Soocho ),( Yeun Jun Chung 대한내과학회 2014 대한내과학회 추계학술대회 Vol.2014 No.1
Background: Copy number variations (CNVs) have been implicated in human diseases. However, it remains unclear how they affect immune dysfunction and autoimmune diseases, including rheumatoid arthritis (RA). Methods: To de. ne CNVs, we used SNP genotyping data from the 500 discovery set. The Lsp1 plasmid DNA was tagged with GFP and was then transfected into Jurkat cells. Mice genetically de. cient in Lsp1 (Lsp1 ./. mice) were induced of delayed-type hypersensitivity and antigen-induced arthritis. Results: Here, we identi. ed a novel Lsp1 deletion variant for RA susceptibility. Differentially expressed genes in Lsp1-de. cient primary T cells represent cell motility and immune and cytokine responses. Functional assays demonstrated that LSP1, induced by T cell receptor activation, negatively regulates T cell migration by hampering ERK activation in vitro. In mice with T cell-dependent chronic in. ammation, loss of Lsp1 promotes migration of T cells into the target tissues as well as draining lymph nodes, exacerbating disease severity. Moreover, RA patients show diminished expression of LSP1 in peripheral T cells with increased migratory capacity. Conclusions: Our data highlights the importance of Lsp1 CNVs in the pathogenesis of immune diseases and provides novel insights into the mechanisms underlying T cell migration toward the in. amed synovium in RA.