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Effects of pairing correlations on the neutron skin thickness and the symmetry energy
Choi, Soonchul,Zhang, Ying,Cheoun, Myung-Ki,Kwon, Youngshin,Kim, Kyungsik,Kim, Hungchong American Physical Society 2017 Physical Review C Vol.96 No.2
<P>We investigated effects of pairing correlations on the neutron skin thickness and the symmetry energy of finite nuclei. In this calculation we used Hartree-Fock-Bogoliubov method with Skyrme forces and effective pairing interactions. The results have been compared with available experimental data, Hartree-Fock results as well as the predictions by droplet model. Finally, our discussion was extended to study of the pairing interaction in nuclear matter. Roles of isospin T = 0 pairing in the nuclear matter were also discussed.</P>
Jihyun Yun,Taeho Kim,Soonchul Myung,Hyoweon Bang,Inja Lim 대한생리학회-대한약리학회 2006 The Korean Journal of Physiology & Pharmacology Vol.10 No.2
Employing electrophysiological and cell proliferation assay techniques, we studied the effects of Ca<SUP>2</SUP>- activated K<SUP></SUP> channel modulators on the proliferation of human dermal fibroblasts, which is important in wound healing. Macroscopic voltage-dependent outward K<SUP></SUP> currents were found at about 40 mV stepped from a holding potential of 70 mV. The amplitude of K<SUP></SUP> current was increased by NS1619, a specific large-conductance Ca<SUP>2</SUP>-activated K<SUP></SUP> (BK) channel activator, but decreased by iberiotoxin (IBTX), a specific BK channel inhibitor. To investigate the presence of an intermediate-conductance Ca<SUP>2</SUP>-activated K<SUP></SUP> (IK) channels, we pretreated the fibroblasts with low dose of TEA to block BK currents, and added 1-EBIO (an IK activator). 1-EBIO recovered the currents inhibited by TEA. When various Ca<SUP>2</SUP>-activated K<SUP></SUP> channel modulators were added into culture media for 1∼3 days, NS1619 or 1-EBIO inhibited the cell proliferation. On the other hand, IBTX, clotrimazole or apamin, a small conductance Ca<SUP>2</SUP>-activated K<SUP></SUP> channel (SK) inhibitor, increased it. These results suggest that BK, IK, and SK channels might be involved in the proliferation of human dermal fibroblasts, which is inversely related to the channel activation.
Inja Lim,Jihyun Yun,Seungtae Kim,Soonchul Myung,Taeho Kim,Hyoweon Bang 대한생리학회-대한약리학회 2004 The Korean Journal of Physiology & Pharmacology Vol.13 No.1
Exogenous carbon monoxide (0.2%) increases L-type calcium (Ca<SUP>2</SUP>) current in human jejunal circular smooth muscle cells. The stimulatory effect of carbon monoxide (CO) on L-type Ca<SUP>2</SUP> current is inhibited by pre-application of L-NNA, a classical competitive inhibitor of nitric oxide synthase (NOS) with no significant isoform selectivity (Lim, 2003). In the present study, we investigated which isoform of NOS affected CO induced stimulation of L-type Ca<SUP>2</SUP> current in human jejunal circular smooth muscle cells. Cells were voltage clamped by whole-cell mode patch clamp technique, and membrane currents were recorded with 10 mM barium as the charge carrier. Before the addition of CO, cells were pretreated with each inhibitor of three NOS isoforms for 15 minutes. CO-stimulating effect on L-type Ca<SUP>2</SUP> current was partially blocked by N-(3-(Amino-methyl) benzyl) acetamidine·2HCl (1400W, an iNOS inhibitor). On the other hand, 3-bromo-7-nitroindazole (BNI, a nNOS inhibitor) or N<SUP>5</SUP>-(1-Iminoethyl)-L-ornithine dihydrochloride (L-NIO, an eNOS inhibitor) completely blocked the CO effect. These data suggest that low dose of exogenous CO may stimulate all NOS isoforms to increase L-type Ca<SUP>2</SUP> channel through nitric oxide (NO) pathway in human jejunal circular smooth muscle cells.