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- 검색키워드
- 저자 : Meiling Tang (검색결과 3 건)
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Yong Tang,Meiling Zhong,Hongyao Qin,Yuxuan Liua,Lingxiao Xiang 한국마케팅과학회 2019 마케팅과학연구 Vol.29 No.3
This study seeks to report international tourists’ negative accounts of their visits to Sichuan, anemerging international tourist destination in the southwest of China and the hometownof Giant Pandas. Using the Interpretative Netnography Approach, the study employs the Atlas.ti and NVivo to analyze blog narratives collected from the TravelPod, one of the first website to offer free service to users to create travel blogs. The results indicate that unpleasant encounters with squat toilet became the targets of frequent complaints while the richest accounts were centered on tough and stuffy long bus ride threatened by potential risk of bad road, uncomfortable train tour, and unpleasant air travel experiences. This study recognizes that international travelers’ negative accounts on foreign lands as indicated in travel blogs have been produced in the context of cultural politics, filters and barriers, post-colonialism, and narrative turn, as well as personal point of reference and travelers’ bias. The findings will deepen the current understanding of negative word of mouth narrated in travel blogs so as to respond accordingly and prevent its undesirable consequences to destination image throughout postmodern consumer culture in the economic and cultural globalized world.
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Xingjun Gao,Xiang Li,Yan He,Lin Fan,Meiling Tang 한국정밀공학회 2022 International Journal of Precision Engineering and Vol.23 No.11
In order to solve the problems of low efficiency, pollution and large damage of existing polishing methods, the electrical enhanced photocatalysis polishing method for single-crystal silicon carbide was proposed in this paper. The recombination of electrons and holes was inhibited by the combined action of applied electric field and chemical trapping agent, so the efficiency of oxidation was improved, and the polishing of silicon carbide with high efficiency and low damage can be achieved. The effects of various parameters such as polishing pressure, polishing speed, polishing pad, abrasive particles and oxygenation of slurry on polishing effect were studied. 0.025 MPa of polishing pressure, 60 r/min of polishing speed, 5 wt% of SiO2 abrasive particles and synthetic fiber polymer polishing pad can provide the higher MRR of 1.18 μm/h and the best surface quality (Ra = 0.348 nm). In addition, it has been proven that UV, optimized pH, types of photocatalysts, electron trapping agent and voltage are necessary factors for the slurry to exhibit strong oxidizing properties. The MRR is mainly determined by both mechanical action (introduced by abrasive particles) and chemical action (introduced by slurry). The above technology can basically meet the requirements of high-efficiency, ultra-smooth and low damage polishing of single-crystal silicon carbide.
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Yue Yifeng,Zong Liwu,Chen Yongmin,Nianhai Feng,Tang Junxia,Xu Hongyu,Zhao Meiling 대한독성 유전단백체 학회 2021 Molecular & cellular toxicology Vol.17 No.4
Background Inflammation and oxidative stress-induced molecular death are one of the important causes for lung injury in critically ill patients. LKB1 is an important protein kinase in the body and has regulated inflammation and oxidative stress. However, LKB1 control inflammation and oxidative stress in lung injury were unclear. Objective We aimed to investigate the function and mechanism of Liver kinase B1 (LKB1) in lipopolysaccharide (LPS)-induced lung injury. Result LKB1 prevented lung injury, and weakened correlation oxidative stress and inflammatory reaction in LPS-induced mice model of lung injury. Up-regulation of LKB1 reduced reactive oxygen species (ROS) production and it-induced oxidative stress, and weakened inflammatory reactions in LPS-induced lung injury A549 cells. Down-regulation of LKB1 increased ROS production and it-induced oxidative stress, and enhanced inflammatory reactions in LPS-induced lung injury A549 cells. LKB1 induced phosphorylation (p)-AMPK protein expression, and suppressed the protein expression of NLRP3 in LPS-induced mice model of lung injury and in LPS-induced lung injury A549 cells. This experiment demonstrated the inhibition of AMPK or activation of NLRP3 inflammasome reversed the anti-inflammation function of LKB1 in LPS-induced lung injury. Meanwhile, ROS-induced oxidative stress also participated in the anti-inflammation effects of LKB1 in LPS-induced lung injury. Conclusion Therefore, our results indicate that LKB1 reduced inflammation and oxidative stress by regulating the AMPK/NLRP3 signaling pathway in LPS-induced lung injury.
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