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Simulation and Optimization Study on Layout Planning of Plant Factory Based on WITNESS
Li Ma,Meiqiong Ma,Chao Ma,Jingwen Deng,Xingluo Liu,Lijun Zhao 보안공학연구지원센터 2016 International Journal of Security and Its Applicat Vol.10 No.5
The emergence of plant factory has solved great restriction problem of natural environment in the traditional agriculture. Therefore, it has been the research concentrate on the facility agriculture in recent years. However, the reasonability of factory's production process and layout determines quality of the product, production efficiency and economic benefits. This paper took Wuchang Jingtian Plant Factory as an example, analyzed the production process and layout and provided a corresponding improvement scheme which was verified by the WITNESS simulation software. An optimal solution was determined from the evaluation index of production efficiency, busy rate, production cycle etc.. Besides, the simulation analysis of production cost before and after the improvement verified the improved plant factory operation is effective and feasible. This improvement scheme not only improved efficiency of production system of the plant factory, but also reduced the operating cost.
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Rongyi Zhang,Junhua Wang,Qingwen Deng,Xingru Xiao,Xiang Zeng,Biqin Lai,Ge Li,Yuanhuan Ma,Jingwen Ruan,한인보,Yuan-Shan Zeng,Ying Ding 대한척추신경외과학회 2023 Neurospine Vol.20 No.4
Objective: Herein, we investigated whether mesenchymal stem cells (MSCs) transplantation combined with electroacupuncture (EA) treatment could decrease the proportion of proinflammatory microglia/macrophages and neurotoxic A1 reactive astrocytes and inhibit glial scar formation to enhance axonal regeneration after spinal cord injury (SCI). Methods: Adult rats were divided into 5 groups after complete transection of the spinal cord at the T10 level: a control group, a nonacupoint EA (NA-EA) group, an EA group, an MSC group, and an MSCs+EA group. Immunofluorescence labeling, quantitative real-time polymerase chain reaction, enzyme-linked immunosorbent assay, and Western blots were performed. Results: The results showed that MSCs+EA treatment reduced the proportion of proinflammatory M1 subtype microglia/macrophages, but increased the differentiation of anti-inflammatory M2 phenotype cells, thereby suppressing the mRNA and protein expression of proinflammatory cytokines (tumor necrosis factor-α and IL-1β) and increasing the expression of an anti-inflammatory cytokine (interleukin [IL]-10) on days 7 and 14 after SCI. The changes in expression correlated with the attenuated neurotoxic A1 reactive astrocytes and glial scar, which in turn facilitated the axonal regeneration of the injured spinal cord. In vitro, the proinflammatory cytokines increased the level of proliferation of astrocytes and increased the expression levels of C3, glial fibrillary acidic protein, and chondroitin sulfate proteoglycan. These effects were blocked by administering inhibitors of ErbB1 and signal transducer and activator of transcription 3 (STAT3) (AG1478 and AG490) and IL-10. Conclusion: These findings showed that MSCs+EA treatment synergistically regulated the microglia/macrophage subpopulation to reduce inflammation, the formation of neurotoxic A1 astrocytes, and glial scars. This was achieved by downregulating the ErbB1-STAT3 signal pathway, thereby providing a favorable microenvironment conducive to axonal regeneration after SCI.
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