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RISS 인기검색어
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- 저자 : ( Yibin Tang ) (검색결과 3 건)
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Spectrum Hole Utilization in Cognitive Two-way Relaying Networks
( Yuan Gao ),( Changping Zhu ),( Yibin Tang ) 한국인터넷정보학회 2014 KSII Transactions on Internet and Information Syst Vol.8 No.3
This paper investigates the spectrum hole utilization of cooperative schemes for the two-way relaying model in order to improve the utilization efficiency of limited spectrum holes in cognitive radio networks with imperfect spectrum sensing. We propose two specific bidirectional secondary data transmission (BSDT) schemes with two-step and three-step two-way relaying models, i.e., two-BSDT and three-BSDT schemes, where the spectrum sensing and the secondary data transmission are jointly designed. In the proposed cooperative schemes, the best two-way relay channel between two secondary users is selected from a group of secondary users serving as cognitive relays and assists the bi-directional communication between the two secondary users without a direct link. The closed-form asymptotic expressions for outage probabilities of the two schemes are derived with a primary user protection constraint over Rayleigh fading channels. Based on the derived outage probabilities, the spectrum hole utilization is calculated to evaluate the percentage of spectrum holes used by the two secondary users for their successful information exchange without channel outage. Numerical results show that the spectrum hole utilization depends on the spectrum sensing overhead and the channel gain from a primary user to secondary users. Additionally, we compare the spectrum hole utilization of the two schemes as the varying of secondary signal to noise ratio, the number of cognitive relays, and symmetric and asymmetric channels.
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Adaptive Cooperation for Bidirectional Communication in Cognitive Radio Networks
( Yuan Gao ),( Changping Zhu ),( Zhixiang Deng ),( Yibin Tang ) 한국인터넷정보학회 2017 KSII Transactions on Internet and Information Syst Vol.11 No.3
In the interweave cognitive networks, the interference from the primary user degrades the performance of the cognitive user transmissions. In this paper, we propose an adaptive cooperation scheme in the interweave cognitive networks to improve the performance of the cognitive user transmissions. In the proposed scheme for the bidirectional communication of two end-source cognitive users, the bidirectional communication is completed through the non-relay direct transmission, the one-way relaying cooperation transmission, and the two-way relaying cooperation transmission depending on the limited feedback from the end-sources. For the performance analysis of the proposed scheme, we derive the outage probability and the finite-SNR diversity multiplexing tradeoff (f-DMT) in a closed form, considering the imperfect spectrum sensing, the interference from the primary user, and the power allocation between the relay and the end-sources. The results show that compared with the direct transmissions (DT), the pure one-way relaying transmissions (POWRT), and the pure two-way relaying transmissions (PTWRT), the proposed scheme has better outage performance. In terms of the f-DMT, the proposed scheme outperforms the full cooperation transmissions of the POWRT and PTWRT.
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RhoGDI2 induced malignant phenotypes of pancreatic cancer cells via regulating Snail expression
Yi Bin,Hu You,Zhu Dongming,Yao Jun,Zhou Jian,Zhang Yi,He Zhilong,Zhang Lifeng,Zhang Zixiang,Yang Jian,Tang Yuchen,Huang Yujie,Li Dechun,Liu Qiuhua 한국유전학회 2022 Genes & Genomics Vol.44 No.5
Background: Rho GDP dissociation inhibitor 2 (RhoGDI2) has been shown to contribute to the aggressive phenotypes of human cancers, such as tumor metastasis and chemoresistance. Objective: This study aimed to assess the effects of RhoGDI2 on tumor progression and chemoresistance in pancreatic cancer cells. Methods: The expression of RhoGDI2 in pancreatic cancer cells was detected by Western blot analysis. Gain-of-function and loss-of-function approaches were done to examine the malignant phenotypes of the RhoGDI2-expressing or RhoGDI2-depleting cells. The correlation between RhoGDI2 and Snail was also analyzed. Results: Differential expression of RhoGDI2 protein in pancreatic cancer cell lines was identified. Gain-of-function and loss-of-function experiments showed that RhoGDI2 induced the malignant phenotypes of pancreatic cancer cells, including proliferation, migration, invasion, and gemcitabine (GEM) chemoresistance. The upregulation of RhoGDI2 stimulated the expression of Snail, resulting in the altered expression of epithelial marker E-cadherin and mesenchymal marker Vimentin, which were characteristics of the tumorigenic activity of epithelial-mesenchymal transition. The expression of RhoGDI2 and Snail was upregulated in clinical tumor samples, and higher expression of RhoGDI2 or Snail was significantly associated with poor patient survival in pancreatic ductal adenocarcinoma (PDAC). Conclusion: The findings indicated that RhoGDI2 promoted GEM resistance and tumor progression in pancreatic cancer and that RhoGDI2 might be a potential therapeutic target in patients with PDAC.
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