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Active Muscle and Whole Body Lactate Kinetics after Endurance Training in Men
( Bryan C. Bergman ),( Eugene E. Wolfel ),( Gail E. Butterfield ),( Gary D. Lopaschuk ),( Gretchen A. Casazza ),( Michael A. Horning ),( George A. Brooks ) 한국스포츠정책과학원(구 한국스포츠개발원) 1999 International Journal of Applied Sports Sciences Vol.11 No.1
We evaluated the hypotheses that endurance training decreases arterial lactate concentration ([lactate]a) during continuous exer-cise by decreasing net lactate release (L) and appearance rates (Ra) and increasing metabolic clearance rate (MCR). Measurements were made at two intensities before [45 and 65% peak O2 consumption (VO2peak)] and after training [65% pretraining VO2peak, same absolute workload (ABT), and 65% posttraining VO2peak, same relative intensity (RLT)]. Nine men (27.4 2.0 yr) trained for 9 wk on a cycle ergometer, 5 times/wk at 75% VO2peak, Compared with the 65% VO2peak:, pretraining condition (4.75 0.4 mM), [lactate]a decreased at ABT (41%) and RLT (21%) (P,0.05). L decreased at ABT but not at RLT. Leg lactate uptake and oxidation were unchanged at ABT but increased at RLT. MCR was unchanged at ABT but increased at RLT. We conclude that 1) active skeletal muscle is not solely responsible for elevated [lactate]a; and 2) training increases leg lactate clearance, decreases whole body and leg lactate production at a given moderate-intensity power output, and increases both whole body and leg lactate clearance at a high relative power output.