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Computer Network Vulnerability Assessment and Safety Evaluation Application based on Bayesian Theory
Liang Huang,Xinhao Chen,Xinsheng Lai 보안공학연구지원센터 2016 International Journal of Security and Its Applicat Vol.10 No.12
Computer network vulnerability analysis is a method of analysis and evaluation of network security beforehand. The attacks method has occurred in the network, the previous network status change as input information, calculated by the model analysis. Forecasting network node may be network attacks given the current security level value network, network security reinforcement measures taken before the danger. Administrators can proactively identify network security issues, to take measures in advance to avoid information leakage, financial losses, ensure the safety of individuals and countries. Therefore, vulnerability analysis computer network is very important. Based on the properties of attack graph shows the method of attack graphs to Bayesian network transformation, using the new algorithm to eliminate loops attribute attack graph optimization, building the Bayesian attribute attack graph model used to evaluate the network itself security situation. In this model, based on Bayes formula for calculating the probability of a new node probability calculation formula and attack paths occur for calculating network vulnerability assessment of the quantitative indicators. The model not only can visually process description of cyber attacks, but also into the Bayesian network probabilistic thinking of possible network attack path prediction and assessment.
Huang, Xi,Ouyang, Xinhao,Yang, Panyu,Lau, On Sun,Chen, Liangbi,Wei, Ning,Deng, Xing Wang National Academy of Sciences 2013 PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF Vol.110 No.41
<P>The evolutionarily conserved CONSTITUTIVE PHOTOMORPHOGENESIS 1 (COP1) is a RING and WD40 protein that functions as a substrate receptor of CULLIN4–DAMAGED DNA BINDING PROTEIN 1 (CUL4–DDB1)–based E3 ubiquitin ligases in both plants and animals. In <I>Arabidopsis</I>, COP1 is a central repressor of photomorphogenesis in the form of COP1–SUPPRESSOR OF PHYA (SPA) complex(es). CUL4–DDB1–COP1–SPA suppresses the photomorphogenic program by targeting the transcription factor ELONGATED HYPOCOTYL 5 for degradation. Intriguingly, under photomorphogenic UV-B light, COP1 reverses its repressive role and promotes photomorphogenesis. However, the mechanism by which COP1 is functionally switched is still obscure. Here, we demonstrate that UV-B triggers the physical and functional disassociation of the COP1–SPA core complex(es) from CUL4–DDB1 and the formation of a unique complex(es) containing the UV-B receptor UV RESISTANCE LOCUS 8 (UVR8). The establishment of this UV-B–dependent COP1 complex(es) is associated with its positive modulation of ELONGATED HYPOCOTYL 5 stability and activity, which sheds light on the mechanism of COP1’s promotive action in UV-B–induced photomorphogenesis.</P>