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Yongqiang Zhang,Xiangyi Xue,Jingli Zhang,Shewei Xin,Hao Pan,Huamei Sun,Huiming Li 대한금속·재료학회 2021 METALS AND MATERIALS International Vol.27 No.7
The hot deformation behavior of Ti–22Al–25Nb was studied by the high temperature compression over a range of temperatures (950–1050 °C) and strain rates (0.001–10 s−1) in this paper. The work-hardening (WH) and softening deformationbehaviors of Ti–22Al–25Nb were analyzed. Obvious linear decreasing regimes of WH rate curves can be found before thedynamic recrystallization (DRX) onset, which indicates WH+ DRV (dynamic recovery) stage. And WH rate decreasedsignifcantly with strain rate reduced and temperature elevated. A physically-based constitutive model was established,which can well predict the fow behavior of Ti–22Al–25Nb. Additional, strain-rate sensitivity coefcient distribution mapwas established. The higher values of m appeared at the low strain rate. When the strain rate exceeded 0.1 s−1, the values ofm were lower than 0.25.
Complex dental anomalies in a belatedly diagnosed cleidocranial dysplasia patient
Lu, Hui,Zeng, Binghui,Yu, Dongsheng,Jing, Xiangyi,Hu, Bin,Zhao, Wei,Wang, Yiming Korean Academy of Oral and Maxillofacial Radiology 2015 Imaging Science in Dentistry Vol.46 No.1
Cleidocranial dysplasia (CCD) is a rare congenital disorder, typically characterized by persistently open skull sutures, aplastic or hypoplastic clavicles, and supernumerary teeth. Mutations in the gene encoding the runt-related transcription factor 2 (RUNX2) protein are responsible for approximately two thirds of CCD patients. We report a 20-year-old CCD patient presenting not only with typical skeletal changes, but also complex dental anomalies. A previously undiagnosed odontoma, 14 supernumerary teeth, a cystic lesion, and previously unreported fused primary teeth were discovered on cone-beam computed tomography (CBCT) scans. Mutation analysis identified the causal c.578G>A (p.R193Q) mutation in the RUNX2 gene. At 20 years of age, the patient had already missed the optimal period for dental intervention. This report describes the complex dental anomalies in a belatedly diagnosed CCD patient, and emphasizes the significance of CBCT assessment for the detection of dental anomalies and the importance of early treatment to achieve good outcomes.
Complex dental anomalies in a belatedly diagnosed cleidocranial dysplasia patient
Hui Lu,Binghui Zeng,Dongsheng Yu,Xiangyi Jing,Bin Hu,Wei Zhao,Yiming Wang 대한영상치의학회 2015 Imaging Science in Dentistry Vol.45 No.3
Cleidocranial dysplasia (CCD) is a rare congenital disorder, typically characterized by persistently open skull sutures, aplastic or hypoplastic clavicles, and supernumerary teeth. Mutations in the gene encoding the runt-related transcription factor 2 (RUNX2) protein are responsible for approximately two thirds of CCD patients. We report a 20-year-old CCD patient presenting not only with typical skeletal changes, but also complex dental anomalies. A previously undiagnosed odontoma, 14 supernumerary teeth, a cystic lesion, and previously unreported fused primary teeth were discovered on cone-beam computed tomography (CBCT) scans. Mutation analysis identified the causal c.578G>A (p.R193Q) mutation in the RUNX2 gene. At 20 years of age, the patient had already missed the optimal period for dental intervention. This report describes the complex dental anomalies in a belatedly diagnosed CCD patient, and emphasizes the significance of CBCT assessment for the detection of dental anomalies and the importance of early treatment to achieve good outcomes.