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DETERMINATION OF FUNCTIONALITY DISTRIBUTIONS IN TELECHELIC PREPOLYMERS BY THIN-LAYER CHROMATOGRARHY
Min, T.I.,T. MIYAMOTO,H. INAGAKI 亞州大學校 1978 論文集 Vol.2 No.-
Functionality distributions of telechelic prepolymers have been determined by thin layer chromatography (TLC). Commercially available 1,2-poly-butadienes having either carboxyl or hydroxyl groups were examined. TLC with p-xylene as the developer made it possible to separate the sample into a nonfunctional component and a mixture of mono-and difunctional componenets. Complete separation of the sample into the three components was achieved by selecting the developer and development procedure appropriately. Quantification of the chromatograms was performed successfully in a TLC apparatus equipped with a flame ionization detector. A simple method for determination of the functionality distribution was proposed.
Obi, T.,Matsumoto, M.,Miyazaki, K.,Kitsutaka, K.,Tamaki, M.,Takase, K.,Miyamoto, A.,Oka, T.,Kawamoto, Y.,Nakada, T. Asian Australasian Association of Animal Productio 2010 Animal Bioscience Vol.23 No.9
Comparisons of properties between skeletal ryanodine receptor 1 (sRyR1)-heterozygous-mutated and normal types of meat were carried out in pigs using PSE (pale, soft and exudative) meat found during the butchering process. All samples considered to be PSE meat showed irregular running and disorder of the muscle fibers and a wider inter-fiber space upon light microscopic observation. Electron microscopy revealed disintegration, twisting, and disorder of the myofibril arrangement and elimination of the Z line in PSE meat, compared with normal meat. Meat property tests demonstrated greater decreases in water holding capacity, moisture and sarcoplasmic protein, and higher $L^*$ values for the meat color index in PSE meat than in normal meat, but there were no differences in these factors between genetically normal and sRyR1-heterozygous PSE meat. On the other hand, higher $a^*$ and $b^*$ values were observed in sRyR1-heterozygous than in normal PSE meat, and similar alterations to the a* value were observed in terms of the amount of myoglobin and density of the 17-kDa protein band, corresponding to the molecular mass of myoglobin, on SDS-PAGE gels. These results suggest that sRyR1-heterozygous PSE pork contains much more myoglobin than genetically normal PSE meat.
Tuning of magnetic and transport properties in Bi2Te3by divalent Fe doping
Jo, N. H.,Lee, K. J.,Kim, C. M.,Okamoto, K.,Kimura, A.,Miyamoto, K.,Okuda, T.,Kim, Y. K.,Lee, Z.,Onimaru, T.,Takabatake, T.,Jung, M. H. American Physical Society 2013 Physical review. B, Condensed matter and materials Vol.87 No.20
Oxidative Stress Interferes With White Matter Renewal After Prolonged Cerebral Hypoperfusion in Mice
Miyamoto, Nobukazu,Maki, Takakuni,Pham, Loc-Duyen D.,Hayakawa, Kazuhide,Seo, Ji Hae,Mandeville, Emiri T.,Mandeville, Joseph B.,Kim, Kyu-Won,Lo, Eng H.,Arai, Ken American Heart Association, Inc. 2013 Stroke Vol.44 No.12
<P><B>Background and Purpose—</B></P><P>White matter injury caused by cerebral hypoperfusion may contribute to the pathophysiology of vascular dementia and stroke, but the underlying mechanisms remain to be fully defined. Here, we test the hypothesis that oxidative stress interferes with endogenous white matter repair by disrupting renewal processes mediated by oligodendrocyte precursor cells (OPCs).</P><P><B>Methods—</B></P><P>In vitro, primary rat OPCs were exposed to sublethal CoCl<SUB>2</SUB> for 7 days to induce prolonged chemical hypoxic stress. Then, OPC proliferation/differentiation was assessed. In vivo, prolonged cerebral hypoperfusion was induced by bilateral common carotid artery stenosis in mice. Then, reactive oxygen species production, myelin density, oligodendrocyte versus OPC counts, and cognitive function were evaluated. To block oxidative stress, OPCs and mice were treated with the radical scavenger edaravone.</P><P><B>Results—</B></P><P>Prolonged chemical hypoxic stress suppressed OPC differentiation in vitro. Radical scavenging with edaravone ameliorated these effects. After 28 days of cerebral hypoperfusion in vivo, reactive oxygen species levels were increased in damaged white matter, along with the suppression of OPC-to-oligodendrocyte differentiation and loss of myelin staining. Concomitantly, mice showed functional deficits in working memory. Radical scavenging with edaravone rescued OPC differentiation, ameliorated myelin loss, and restored working memory function.</P><P><B>Conclusions—</B></P><P>Our proof-of-concept study demonstrates that after prolonged cerebral hypoperfusion, oxidative stress interferes with white matter repair by disrupting OPC renewal mechanisms. Radical scavengers may provide a potential therapeutic approach for white matter injury in vascular dementia and stroke.</P>