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Implementation of Biometric Techniques in Social Networking Sites
Shilpi Sharma,J. S. Sodhi 보안공학연구지원센터 2014 International Journal of Security and Its Applicat Vol.8 No.6
With the advancement of technologyprivacy threats arise while establishing communication in social networking sites. For the developers of authentication systems user’s privacy and authentication have evolved as a major problem and area of concern. This research is based on an attempt to discuss the implementation of different biometric verification techniques in order to perform the authentication of users in social networking sites. It decreases the chances of illegal impersonation thus enhancing the privacy of an individual’s personal data. The prime objective of this paper is to provide a new revolution in social networking sites by suggesting remedial measures for the security threats faced in the sites such as Facebook, MySpace, Twitter, Hi5, LinkedIn, Orkut, Tumbler, Flikeretc so that the goal of communication can be achieved without any security threat in mind. Application of securedbiometricsenables confirmation of personal detailsfor establishing the identity of an individual.
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Kirtisudha Mishra,Shilpy Singla,Suvasini Sharma,Renu Saxena,Vineeta Vijay Batra 대한소아청소년과학회 2014 Clinical and Experimental Pediatrics (CEP) Vol.57 No.2
Griscelli syndrome type 2 (GS2) is a rare autosomal recessive disease caused by mutations in the RAB27Agene. It is characterized by cutaneous hypopigmentation, immunodeficiency, and hemophagocyticlymphohistiocytosis. We describe 2 brothers who had GS2 with clinically diverse manifestations. The elderbrother presented with a purely neurological picture, whereas the younger one presented with fever,pancytopenia, hepatosplenomegaly, and erythema nodosum. Considering that cutaneous hypopigmentationwas a common feature between the brothers, genetic analysis for Griscelli syndrome wasperformed. As the elder sibling had died, mutation analysis was only performed on the younger sibling,which revealed a novel homozygous mutation in the RAB27A gene on chromosome 15 showing asingle-base substitution (c.136T>A p.F46I). Both parents were heterozygous for the same mutation. This confirmed the diagnosis of GS2 in the accelerated phase in both siblings. The atypical features ofGS2 in these cases are a novel mutation, isolated neurological involvement in one sibling, associationwith erythema nodosum, and 2 distinct clinical presentations in siblings with the same genetic mutation.
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Mishra, Kirtisudha,Singla, Shilpy,Sharma, Suvasini,Saxena, Renu,Batra, Vineeta Vijay The Korean Pediatric Society 2014 Clinical and Experimental Pediatrics (CEP) Vol.57 No.2
Griscelli syndrome type 2 (GS2) is a rare autosomal recessive disease caused by mutations in the RAB27A gene. It is characterized by cutaneous hypopigmentation, immunodeficiency, and hemophagocytic lymphohistiocytosis. We describe 2 brothers who had GS2 with clinically diverse manifestations. The elder brother presented with a purely neurological picture, whereas the younger one presented with fever, pancytopenia, hepatosplenomegaly, and erythema nodosum. Considering that cutaneous hypopigmentation was a common feature between the brothers, genetic analysis for Griscelli syndrome was performed. As the elder sibling had died, mutation analysis was only performed on the younger sibling, which revealed a novel homozygous mutation in the RAB27A gene on chromosome 15 showing a single-base substitution (c.136T>A p.F46I). Both parents were heterozygous for the same mutation. This confirmed the diagnosis of GS2 in the accelerated phase in both siblings. The atypical features of GS2 in these cases are a novel mutation, isolated neurological involvement in one sibling, association with erythema nodosum, and 2 distinct clinical presentations in siblings with the same genetic mutation.
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