http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
Development of foam-based layered targets for laser-driven ion beam production
Prencipe, I,Sgattoni, A,Dellasega, D,Fedeli, L,Cialfi, L,Choi, Il Woo,Kim, I Jong,Janulewicz, K A,Kakolee, K F,Lee, Hwang Woon,Sung, Jae Hee,Lee, Seong Ku,Nam, Chang Hee,Passoni, M IOP 2016 Plasma physics and controlled fusion Vol.58 No.3
<P>We report on the development of foam-based double-layer targets (DLTs) for laser-driven ion acceleration. Foam layers with a density of a few mg cm<SUP>−3</SUP> and controlled thickness in the 8–36 <I>μ</I>m range were grown on <I>μ</I>m-thick Al foils by pulsed laser deposition (PLD). The DLTs were experimentally investigated by varying the pulse intensity, laser polarisation and target properties. Comparing DLTs with simple Al foils, we observed a systematic enhancement of the maximum and average energies and number of accelerated ions. Maximum energies up to 30 MeV for protons and 130 MeV for C<SUP>6+</SUP> ions were detected. Dedicated three-dimensional particle-in-cell (3D-PIC) simulations were performed considering both uniform and cluster-assembled foams to interpret the effect of the foam nanostructure on the acceleration process.</P>
Giovanna Lattanzi,Camilla Pellegrini,Marta Columbaro,Elisa Schena,Sabino Prencipe,Davide Andrenacci,Patricia Iozzo,Maria Angela Guzzardi,Cristina Capanni,Elisabetta Mattioli,Manuela Loi,David Araujo-V 생화학분자생물학회 2019 Experimental and molecular medicine Vol.51 No.-
Type-2 Familial Partial Lipodystrophy is caused by LMNA mutations. Patients gradually lose subcutaneous fat from the limbs, while they accumulate adipose tissue in the face and neck. Several studies have demonstrated that autophagy is involved in the regulation of adipocyte differentiation and the maintenance of the balance between white and brown adipose tissue. We identified deregulation of autophagy in laminopathic preadipocytes before induction of differentiation. Moreover, in differentiating white adipocyte precursors, we observed impairment of large lipid droplet formation, altered regulation of adipose tissue genes, and expression of the brown adipose tissue marker UCP1. Conversely, in lipodystrophic brown adipocyte precursors induced to differentiate, we noticed activation of autophagy, formation of enlarged lipid droplets typical of white adipocytes, and dysregulation of brown adipose tissue genes. In agreement with these in vitro results indicating conversion of FPLD2 brown preadipocytes toward the white lineage, adipose tissue from FPLD2 patient neck, an area of brown adipogenesis, showed a white phenotype reminiscent of its brown origin. Moreover, in vivo morpho-functional evaluation of fat depots in the neck area of three FPLD2 patients by PET/CT analysis with cold stimulation showed the absence of brown adipose tissue activity. These findings highlight a new pathogenetic mechanism leading to improper fat distribution in lamin A-linked lipodystrophies and show that both impaired white adipocyte turnover and failure of adipose tissue browning contribute to disease.
Measurement of theB+→ωl+νandB+→ηl+νbranching fractions
Aubert, B.,Bona, M.,Karyotakis, Y.,Lees, J. P.,Poireau, V.,Prencipe, E.,Prudent, X.,Tisserand, V.,Garra Tico, J.,Grauges, E.,Lopez, L.,Palano, A.,Pappagallo, M.,Eigen, G.,Stugu, B.,Sun, L.,Abrams, G. American Physical Society 2009 PHYSICAL REVIEW D - Vol.79 No.5