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RISS 인기검색어
- 검색키워드
- 저자 : Li Zheng-bang (검색결과 4 건)
- 무료
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- 유료
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Nonsingular Fast Terminal Sliding Mode Control Strategy for PMLSM Based on Disturbance Compensation
Li Zheng,Zhang Zi-Hao,Wang Jin-Song,Yan Zhi-Bang,Guo Xiao-Qiang,Sun He-Xu 대한전기학회 2024 Journal of Electrical Engineering & Technology Vol.19 No.3
To optimize the anti-interference performance and dynamic response ability of permanent magnet linear synchronous motor (PMLSM) and enhance the overall control performance of the system, the novel nonsingular fast terminal sliding mode controller (NFTSMC) based on disturbance compensation is proposed in this design. To enhance the response speed and stability of the controller, this design introduces the system state variable into the exponential reaching law to make the controller adaptively adjust with the change of the system state, which can reduce the chattering of the system and have a faster response speed. The extended sliding mode disturbance observer is designed to estimate the interference factors in the system, and the controller is feedforward compensated to further improve the anti-interference ability of the PMLSM system. Meanwhile, the control strategy is proved by the Lyapunov stability theory. Simulation and experiments show that the proposed control strategy has better dynamic response ability and anti-interference ability than the traditional NFTSMC.
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Li Zheng,Yan Zhi-Bang,Zhang Lu-Cheng,Liu Yu,Zhang Hong-Jie,Sun He-Xu 대한전기학회 2024 Journal of Electrical Engineering & Technology Vol.19 No.3
In recent years, under the background of low carbonization and industrial intelligence, with the rise of new energy sources such as wind energy and solar energy, rare earth permanent magnets have begun to be widely used in generators of these new energy sources. From the perspective of green and sustainable development, permanent magnet motor can reduce energy loss and meet the requirements of sustainable development through the reasonable selection and utilization of permanent magnet materials, the design of motor structures, and advanced transmission control technology. In this paper, a 225 kW/34,000 rpm high speed permanent magnet motor (HSPMSM) is designed. Firstly, the electromagnetic design of the motor is carried out, and the fnite element model is established. The rotor temperature rise under diferent materials is analyzed by comparing the electromagnetic characteristics and loss of diferent permanent magnet materials and using the principle of electrothermal coupling. Combined with the thermal performance of the material, the advantages of rare earth permanent magnet are analyzed, and SmCo permanent magnet material is selected to be more suitable for the motor. Due to the motor's high speed and high frequency harmonic rate, an improved proportional resonant algorithm based on active disturbance rejection is studied and analyzed. Compared with the traditional PI control, the algorithm further reduces the harmonic content of the motor's stator current while improving the motor's running state, thereby reducing the overall loss of the motor. The prototype and experimental platform are made, and the experimental results verify the correctness of the simulation analysis.
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Silencing of Rac3 Inhibits Proliferation and Induces Apoptosis of Human Lung Cancer Cells
Liu, Tie-Qin,Wang, Ge-Bang,Li, Zheng-Jun,Tong, Xiang-Dong,Liu, Hong-Xu Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.7
Background: Rac3, a member of the Rac family of small guanosine triphosphatases (GTPases), regulates a variety of cell functions, including the organization of the cytoskeleton, cell migration, and invasion. Overexpression of Rac3 has been reported in several human cancers. However, the role of Rac3 in lung cancer (LC) has not been determined in detail. The purpose of this study was to investigate the effect of silencing of Rac3 expression in human LC cells and the consequences for cell survival. Materials and Methods: Lentivirus small hairpin RNA (shRNA) interference techniques were utilized to knock down the Rac3 gene. Gene and protein expression was quantified by quantitative real-time polymerase chain reaction (qRT-PCR) and Western blotting. LC cell apoptosis was examined by annexin V-APC /propidium iodide staining. Results: Efficient silencing of Rac3 strongly inhibited A549 cell proliferation and colony formation ability, and significantly decreased tumor growth. Moreover, flow cytometry analysis showed that knockdown of Rac3 led to G2/M phase cell cycle arrest as well as an excess accumulation of cells in the G1 and S phase. Conclusions: Thus, functional analysis using shRNAs revealed a critical role for Rac3 in the tumor growth of LC cells. shRNA silencing of Rac3 could provide an effective strategy to treat LC.
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