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Jinyan Hu,Dingsheng Wu,Quan Feng,Anfang Wei,Beibei Song 한국섬유공학회 2020 Fibers and polymers Vol.21 No.8
Currently, the preparation of novel efficient and recyclable photocatalysts for pollutant degradation in water hasbecome a research hotpot. In this study, a TiO2-loaded non-woven polypropylene/bacterial cellulose (TiO2-loaded NPBC)composite film was prepared by a biological culturing method. The TiO2 nanoparticles were spontaneously embedded intothe prepared composite film. Then, the composite film was used to degrade methylene blue (MB) and its degradation effectswere explored. The morphology of the composite film was investigated. It was clear that a large amount of TiO2nanoparticles were embedded into the composite membrane during the biological culturing method. The experimental resultsconfirmed that the TiO2-loaded NPBC composite film had good degradation performance and reusability. When the reactiontime was 120 min, the removal rate of MB by the film was 92.8 %. The removal rate remained above 85 % after 5degradation cycles.
Junjun Mao,Juntian Liu,Xiaoming Pang,Ming Li,Jinyan Song,Chunjie Han,Di Wu,Shuyue Wang 한국분자세포생물학회 2012 Molecules and cells Vol.34 No.5
Atherosclerosis is an inflammatory disease in the vessel wall. Nicotine, a major component of cigarette smoke, is an independent risk factor for cardiovascular diseases including atherosclerosis. As an inflammatory molecule, C- reactive protein (CRP) participates in atherogenesis. Although it has been confirmed that CRP level in smoking patient is significantly higher than non-smokers and cigarette withdrawal,it is unknown whether nicotine induces CRP expression in macrophages. The present study was to observe effect of nicotine on CRP production and the related signal pathway in U937 macrophages. The results showed that nicotine significantly increased mRNA and protein expression of CRP in U937 macrophages in time- and concentration-dependent ways. Nicotinic acetylcholine receptor (nAChR) blocker hexamethonium, MEK1/2 inhibitor PD98059, p38 MAPK inhibitor SB203580 and NF-B inhibitor PDTC almost completely abolished nicotine-induced CRP expression in mRNA and protein levels in U937 macrophages. The further study indicated that hexa-methonium, PD98059, and SB203580 significantly inhibited ERK1/2 and p38 MAPK phosphorylation. These demonstrate that nicotine has ability to induce CRP ex-pression in macrophages through nAChR-ERK1/2/p38 MAPK-NF-B signal pathway, which contributes to better understanding of the pro-inflammatory and pro-atherosclerotic effects of nicotine in cigarette smokers.