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Damage Mechanism of Broad-narrow Joint of CRTSII Slab Track under Temperature Rise
Xiaokai Liu,Wenhao Zhang,Jieling Xiao,Xueyi Liu,Wei Li 대한토목학회 2019 KSCE JOURNAL OF CIVIL ENGINEERING Vol.23 No.5
There is two typical damage pattern at broad-narrow joint of CRTSII slab track breakage of narrow joint and fracture at junction between broad joint and narrow joint. This paper aims to study the damage mechanism and develop of broad-narrow joint of CRTSII slab track and put forward the methods to reduce the damage. Based on damaged plasticity model for concrete and cohesive zone model, the damage mechanism and development process of broad-narrow joint are analyzed in this study. The unequal width of broad joint and narrow joint, different concrete strength between broad-narrow joints and slab and slab integrity reducing due to the interface are the main reason for the typical damage. It is suggested to set the same width of broad joint and narrow joint, set the same strength of broad-narrow joint and slab and enhance the integrity of the slab by chipping or adding adhesives, among which the former two methods are more effective. Breakage of narrow joint is a gradual compression damage due to the lower concrete strength than slab. Fracture at the junction between narrow joint and broad joint is a mutational tension damage due to the unequal width of broad joint and narrow joint. The temperature gradient has significant effect on the compression damage, but small to the tension damage, it has the risk of complete destruction under extreme conditions.
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Ansel Shao Pin Tang,Kai En Chan,Jingxuan Quek,Jieling Xiao,Phoebe Tay,Margaret Teng,Keng Siang Lee,Snow Yunni Lin,May Zin Myint,Benjamin Tan,Vijay K Sharma,Darren Jun Hao Tan,Wen Hui Lim,Apichat Kaewd 대한간학회 2022 Clinical and Molecular Hepatology(대한간학회지) Vol.28 No.3
Background/Aims: Non-alcoholic fatty liver disease (NAFLD) is associated with the development of cardiovascular disease. While existing studies have examined cardiac remodeling in NAFLD, there has been less emphasis on the development of carotid atherosclerosis and stroke. We sought to conduct a meta-analysis to quantify the prevalence, risk factors, and degree of risk increment of carotid atherosclerosis and stroke in NAFLD. Methods: Embase and Medline were searched for articles relating to NAFLD, carotid atherosclerosis, and stroke. Proportional data was analysed using a generalized linear mixed model. Pairwise meta-analysis was conducted to obtain odds ratio or weighted mean difference for comparison between patients with and without NAFLD. Results: From pooled analysis of 30 studies involving 7,951 patients with NAFLD, 35.02% (95% confidence interval [CI], 27.36–43.53%) had carotid atherosclerosis with an odds ratio of 3.20 (95% CI, 2.37–4.32; P<0.0001). Pooled analysis of 25,839 patients with NAFLD found the prevalence of stroke to be 5.04% (95% CI, 2.74–9.09%) with an odds ratio of 1.88 (95% CI, 1.23–2.88; P=0.02) compared to non-NAFLD. The degree of steatosis assessed by ultrasonography in NAFLD was closely associated with risk of carotid atherosclerosis and stroke. Older age significantly increased the risk of developing carotid atherosclerosis, but not stroke in NAFLD. Conclusions: This meta-analysis shows that a stepwise increment of steatosis of NAFLD can significantly increase the risk of carotid atherosclerosis and stroke development in NAFLD. Patients more than a third sufferred from carotid atherosclerosis and routine assessment of carotid atherosclerosis is quintessential in NAFLD.
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