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      • Research on Smart Campus Based on the Internet of Things and Virtual Reality

        Youliang Huang,Sajid Ali,Xiaoming Bi,Xing Zhai,Renquan Liu,Fengying Guo,Peng Yu 보안공학연구지원센터 2016 International Journal of Smart Home Vol.10 No.12

        Smart campus is an inevitable trend in the development of digital campus construction. With the development of the Internet of Things (IoT) and Virtual Reality (VR) technology, these technologies become the key to the construction of smart campus. The major objective of this study put forward a smart campus system prototype based on Internet technology. This paper firstly introduces the definition and characteristics of the IoT and VR technology. Secondly, presents the architecture and implementation methodology of the system prototype, and analyzes the core idea of smart campus. Finally, discuss the problems should be noticed in the smart campus construction.

      • KCI등재

        Pulmonary IFN-γ Causes Lymphocytic Inflammation and Cough Hypersensitivity by Increasing the Number of IFN-γ-Secreting T Lymphocytes

        Deng Zheng,Ding Wenbin,Li Fengying,Shen Shuirong,Huang Chuqin,Lai Kefang 대한천식알레르기학회 2022 Allergy, Asthma & Immunology Research Vol.14 No.6

        Purpose: Respiratory viral infection increases the number of lung-resident T lymphocytes, which enhance cough sensitivity by producing interferon-γ (IFN-γ). It is poorly understood why IFN-γ-secreting T lymphocytes persist for a long time when the respiratory viruses have been removed. Methods: Repeated pulmonary administration of IFN-γ and intraperitoneal injection with different inhibitors were used to study the effects of pulmonary IFN-γ in mice and guinea pigs. Results: IFN-γ administration caused the increasing of IFN-γ-secreting T lymphocytes in both lung and blood, followed by the elevated physiological level of IFN-γ in the lung, the airway inflammation and the airway epithelial damage. IFN-γ administration also enhanced the cough sensitivity of guinea pigs. IFN-γ activated the STAT1 and extracellular signal-regulated kinase (ERK) pathways in lung tissues, released IFN-γ-inducible protein 10 (IP-10), and resulted in F-actin accumulation in lung-resident lymphocytes. The CXC chemokine receptor 3 (CXCR3) inhibitor potently suppressed all the IFN-γ-induced inflammatory changes. The STAT1 inhibitor mitigated IFN-γ-secreting T lymphocytes infiltration by inhibiting T lymphocytes proliferation. F-actin accumulation and the ERK1/2 pathway contributed to pulmonary IFN-γ-induced augmentation of the airway inflammation and increasing of IFN-γ-secreting T lymphocytes in blood. Conclusions: High physiological levels of IFN-γ in the lung may cause pulmonary lymphocytic inflammation and cough hypersensitivity by increasing the number of IFN-γ-secreting T lymphocytes through the IP-10 and CXCR3 pathways.

      • KCI등재

        CdTe Quantum Dots as Fluorescent Probes for Josamycin Determination

        Jinyun Peng,Keliang Nong,Guangshan Mu,Fengying Huang 대한화학회 2011 Bulletin of the Korean Chemical Society Vol.32 No.8

        A new method for the determination of josamycin has been developed based on quenching of the fluorescence of 3-mercaptopropionic acid-capped CdTe quantum dots (MPA-CdTe QDs) by josamycin in ethanol. Reaction time, interfering substances on the fluorescence quenching, and mechanism of the interaction of CdTe QDs with josamycin were investigated. Under optimum conditions, the relative fluorescence intensity was linearly proportional to the concentration of josamycin between 12.0 and 120.0 μg mL^(−1) with a correlation coefficient of 0.9956 and a detection limit of 2.5 μg mL^(−1). The proposed method was successfully applied to commercial tablets, and the results were satisfactory, i.e. consistent with those of high-performance liquid chromatography (HPLC).

      • SCOPUSKCI등재

        CdTe Quantum Dots as Fluorescent Probes for Josamycin Determination

        Peng, Jinyun,Nong, Keliang,Mu, Guangshan,Huang, Fengying Korean Chemical Society 2011 Bulletin of the Korean Chemical Society Vol.32 No.8

        A new method for the determination of josamycin has been developed based on quenching of the fluorescence of 3-mercaptopropionic acid-capped CdTe quantum dots (MPA-CdTe QDs) by josamycin in ethanol. Reaction time, interfering substances on the fluorescence quenching, and mechanism of the interaction of CdTe QDs with josamycin were investigated. Under optimum conditions, the relative fluorescence intensity was linearly proportional to the concentration of josamycin between 12.0 and 120.0 ${\mu}g\;mL^{-1}$ with a correlation coefficient of 0.9956 and a detection limit of 2.5 ${\mu}g\;mL^{-1}$. The proposed method was successfully applied to commercial tablets, and the results were satisfactory, i.e. consistent with those of high-performance liquid chromatography (HPLC).

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