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간장및 담도 : 복수를 동반한 간경변증 환자에서 혈청 Albumin 및 Aldosterone치의 변화와 Atrial Natriuretic Factor의 대응에 관한 연구
구자영(Ja Young Koo),박병채(Byung Chae Park),서승연(Sung Yeon Suh),한병훈(Byung Hoon Han),이상욱(Sang Wook Lee),서보원(Bo Won Suh),최경희(Kung Hee Choi),남철우(chul Woo Nam) 대한소화기학회 1990 대한소화기학회지 Vol.22 No.2
N/A In order to evaluate the role in 24hr urinary sodium retension of serum albumin, aldosterone and ANF concentration in patients with decompensated liver cirrhosis, we measured the levels of serum albumine, aldosterone, ANF and 24hr urinary sodium excretion in patients with compensated liver cirrhosis (Group I,n=15), decompensated liver cirrhosis (Group II, n=15) and normal control person (Group III, n=13). The results are as follows; 1) Albumin, aldosterone, 24hr urinary sodium output show no defference between group I & III, but albumin and 24 hr urinary sodium output was significanctly decreased (p<0.005), aldosterone was significantly increased (p<0.005) in group II. 2) ANF was 38.0 +- 17.9 fmol/ml in group I, 50.2 +- 33.9 fmol/ml in group II, 23.4 +- 20.3 fmol/ml in group III, showed significant difference between group II and III (p<0.025). 3) Albumine shows positive correlationship with 24 hr urinary sodium output (r=0.642, p<0.005), negative correlationship with ANF (r= -0.346, p<0.025) and aldosterone (r=-0.529, p<0.005). These findings indicate that decreased albumine concentration has a strong relationship with sodium retention and ascites development. ANF begins to increase to compensate the sodium retension at early liver cirrhosis, but not enough at the late stage of liver cirrhosis so brings the ascites, and aldosterone increament is only but a secondary phenomenon to ascites.