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정훈(Hoon Chung),조무길(Moo Gil Cho) 대한구강악안면외과학회 1990 대한구강악안면외과학회지 Vol.16 No.3
Diabetes Mellitus is a metabolic disease developed by the lack of insulin, characterized by an inappropriate elevation of the plasma glucose level and the change in carbohydrate, lipid and protein metabolism. Associated complications include macroangiopathy, microangiopathy, neuropathy and uncontrolled diabetes mellitus enhance the likelihood of virulent bacterial and fungal infection. Therefore ther are need of caution against easy exacerbation of the infection due to the decrease of the host resistance. The authors present the report of the uncontrolled type Ⅱ diabetes mellitus patient who had a mild skin disease initially, but progressed into a severe bilateral sublingual, submental, submandibular abscess, and treated with insulin therapy, incision and drainage, antibiotic therapy, which gave the patient good result.
흰쥐 해마박편에서 veratrine과 고농도 포타슘자극시 칼슘이온이 glutamic acid 유리에 미치는 영향에 관한 연구 : A role of calcium
조무길,김형룡,김관식 대한구강생물학회 1990 International Journal of Oral Biology Vol.14 No.2
Present study was performed to clarify the effect of calcium on the release of glutamic acid employing hippocampal slices. Hippocampal slices(300∼400㎛ thick) were prepared by the method of Kim et al.(1988) and pre-equilibrated in Krebs-bicarbonate medium(KBM, pH 7.4) for 1hr at 37℃. In case of veratrine induced glutamic acid release, pre-equilibrated slices were incubated in fresh KBM and then veratrine(25μM)-containing KBM for 10 min period in the presence or absence of 2.5mM Ca^++. In case of potassium-induced glutamic acid release, pre-equilibrated slices were incubated in fresh KBM and then potassium(50mM)-containing KBM for 5 min period in the presence or absence of 2.5mM Ca^++. Basal and veratrine and potassium-induced release of glutamic acid was determined from recovered medium by HPLC. The observed results were as follows: 1. The release of glutamic acid induced by the 10 min-exposure of 25μM veratrine and 5 min-exposure of 50mM potassium in the presence of 2.5mM Ca^++ was 330.9±25 nmol and 129.6±20.6 nmol, respectively. When compared with released amounts of glutamic acid during the corresponding spontaneous periods, these were 7.8 and 5.1-fold increase respectively. 2. the release of glutamic acid induced by the 10 min-exposure of 25μM veratrine and 5 min-exposure of 50mM potassium in the absence of Ca^++ was 361.8±31.9 nmol and 62.5±7.5 nmol, respectively. When compared with released amounts of glutamic acid during the corresponding spontaneous periods, these were 8.2 and 2.4-fold increase respectively.