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고속철도 환승교통수단의 이미지가 선호도에 미치는 영향 분석
김문령,박상명(Park, Sang-Myeong),이병주,성수련,남궁문(NamGung, Mun) 대한교통학회 2006 대한교통학회 학술대회지 Vol.51 No.-
고속철도는 전국을 ‘1일 생활권’에서 ‘반나절 생활권’으로 단축시키고 사회와 경제 전반의 경쟁력을 제고시키는 새로운 장거리 교통수단으로 자리매김하고 있다. 또한 서울-부산, 목포간의 여행시간을 1시간 이상 단축시킴으로써 장거리 교통수단의 역할을 담당할 것으로 예측하였지만 고속철도 개통이후 수요가 증가하지 않아 수요예측에 대한 문제가 제기되고 있는 실정이다. 고속철도 수요에 영향을 미치는 요인으로는 고속철도의 서비스 요인뿐만 아니라 정차역까지의 접근성 등 환승교통수단에 대한 서비스가 중요한 요인이라고 생각된다.
김문령,김아미,한경덕,배순희,연규월,전이리,홍정애,김아미,배순희,연규월,전이리,조종남,한경덕,홍정애 이화여자대학교 의학회 1972 梨花醫學會誌 Vol.- No.5
With mice, a single intraperioneal injection of toxic dose of carbon tetrachloride is capable of inducing definite degeneration and necrosis of the hepatic calls of the central and midzonal regions of the lobules. The early changes are sinusoidal congestion, hydropic and fatty degeneration at six-twelve hours. In twenty-four hours, the cells of the central and midzonal region undergo complete necrosis which are soon invaded by inflammatory cells, leaving the mesenchymal supporting stroma and sinusoids unaffected. In three days, the necrotic cells in the central zone are disappearing and begin to be replaced by the newly formed hepatic cells. Repair is going on actively, chiefly through mitotic division of the healthy liver cells, not only at the borders of the area of necrosis but diffusely throughout the organ. The protal canals frequently invaded by inflammatory cells. By the seventh day, the dead tissue has removed. Repair is complete by the fourteenth day. There is no evidence of bile duct proliferation.
김문령,김아미,배순희,연규월,전이리,조종남,한경덕,홍정애 이화여자대학교 의과대학 1972 梨花醫學誌 Vol.- No.5
With mice, a single intraperioneal injection of toxic dose of carbon tetrachloride is capable of inducing definite degeneration and necrosis of the hepatic calls of the central and midzonal regions of the lobules. The early changes are sinusoidal congestion, hydropic and fatty degeneration at six-twelve hours. In twenty-four hours, the cells of the central and midzonal region undergo complete necrosis which are soon invaded by inflammatory cells, leaving the mesenchymal supporting stroma and sinusoids unaffected. In three days, the necrotic cells in the central zone are disappearing and begin to be replaced by the newly formed hepatic cells. Repair is going on actively, chiefly through mitotic division of the healthy liver cells, not only at the borders of the area of necrosis but diffusely throughout the organ. The protal canals frequently invaded by inflammatory cells. By the seventh day, the dead tissue has removed. Repair is complete by the fourteenth day. There is no evidence of bile duct proliferation.