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A Coordinated Ciphertext Policy Attribute-based PHR Access Control with User Accountability
( Guofeng Lin ),( Lirong You ),( Bing Hu ),( Hanshu Hong ),( Zhixin Sun ) 한국인터넷정보학회 2018 KSII Transactions on Internet and Information Syst Vol.12 No.4
The personal health record (PHR) system is a promising application that provides precise information and customized services for health care. To flexibly protect sensitive data, attribute-based encryption has been widely applied for PHR access control. However, escrow, exposure and abuse of private keys still hinder its practical application in the PHR system. In this paper, we propose a coordinated ciphertext policy attribute-based access control with user accountability (CCP-ABAC-UA) for the PHR system. Its coordinated mechanism not only effectively prevents the escrow and exposure of private keys but also accurately detects whether key abuse is taking place and identifies the traitor. We claim that CCP-ABAC-UA is a user-side lightweight scheme. Especially for PHR receivers, no bilinear pairing computation is needed to access health records, so the practical mobile PHR system can be realized. By introducing a novel provably secure construction, we prove that it is secure against selectively chosen plaintext attacks. The analysis indicates that CCP-ABAC-UA achieves better performance in terms of security and user-side computational efficiency for a PHR system.
Lei Zhou,Lin Fu,Na Lv,Jing Liu,Yan Li,Xiaosu Chen,Qingyu Xu,Guofeng Chen,Baoxu Pang,Lili Wang,Yonghui Li,Xiaodong Zhang,Li Yu 생화학분자생물학회 2018 Experimental and molecular medicine Vol.50 No.-
The AML1-ETO fusion protein (A/E), which results from the t(8;21) translocation, is considered to be a leukemiainitiating event. Identifying the mechanisms underlying the oncogenic activity of A/E remains a major challenge. In this study, we identified a specific down-regulation of brain acid-soluble protein 1 (BASP1) in t(8;21) acute myeloid leukemia (AML). A/E recognized AML1-binding sites and recruited DNA methyltransferase 3a (DNMT3a) to the BASP1 promoter sequence, which triggered DNA methylation-mediated silencing of BASP1. Ectopic expression of BASP1 inhibited proliferation and the colony-forming ability of A/E-positive AML cell lines and led to apoptosis and cell cycle arrest. The DNMT inhibitor decitabine up-regulated the expression of BASP1 in A/E-positive AML cell lines. In conclusion, our data suggest that BASP1 silencing via promoter methylation may be involved in A/E-mediated leukemogenesis and that BASP1 targeting may be an actionable therapeutic strategy in t(8;21) AML.
( Yunhao Xia ),( Hanshu Hong ),( Guofeng Lin ),( Zhixin Sun ) 한국인터넷정보학회 2017 KSII Transactions on Internet and Information Syst Vol.11 No.9
Cloud computing is a new service to provide dynamic, scalable virtual resource services via the Internet. Cloud market is available to multiple cloud computing resource providers and users communicate with each other and participate in market transactions. However, since cloud computing is facing with more and more security issues, how to complete the allocation process effectively and securely become a problem urgently to be solved. In this paper, we firstly analyze the cloud resource allocation problem and propose a mathematic model based on combinatorial double auction. Secondly, we introduce a trust evaluation mechanism into our model and combine genetic algorithm with simulated annealing algorithm to increase the efficiency and security of cloud service. Finally, by doing the overall simulation, we prove that our model is highly effective in the allocation of cloud resources.