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Zhaohua Chen,Chaohong Dong,Qun Li,Yue Pu,Zhou Lu 한국섬유공학회 2018 Fibers and polymers Vol.19 No.4
α,ω-di[(4-butoxy-piperazin-1-yl)-phosphinic acid methyl ether]-terminated linear polysiloxane (PNPDMS) was synthesized and utilized as the flame retardant and hydrophobing agent. The flame retardance and thermal decomposition behaviors of cotton fabrics were systematically estimated by limiting oxygen index (LOI), thermogravimetric analysis and vertical burning test, respectively. It was found that the LOI of cotton fabric treated with PNPDMS enhanced to 29.82 % compared with cotton fabric without treatment, whose LOI was only 18.00 %. The treated cotton fabric showed a shorter char length, a shorter After-flame time, and no After-glow time as revealed in vertical burning test. The mechanical property in treated cotton fabric was slightly decrease. Furthermore, the grade of water repellency of treated cotton fabric reached to 90 and water contact angle (WCA) increased to 141.90 o compared with cotton fabric without treatment whose WCA was 62.80 o. The result showed that the cotton fabric treated with PNPDMS exhibited excellent flame retardance and hydrophobic properties.
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Le Yang,Xiaojing Zou,Qiansheng Liang,Hao Chen,Jun Feng,Li Yan,Zhaohua Wang,Daixing Zhou,Shusheng Li,Shanglong Yao,Zhi Zheng 생화학분자생물학회 2007 Experimental and molecular medicine Vol.39 No.1
Cardiomyocyte hypertrophy is a major cause of morbidity and mortality worldwide. The aim of this study is to determine the effects of sodium tans - hinone IIA sulfonate (STS) on cardiomyocyte hypertrophy induced by angiotensin II (Ang II) in vivo and in vitro. In long-term treatment, adult Wistar rats were infused with Ang II for three weeks via osmotic mini-pumps and some of them were given intragas - trically of STS. Left ventricle was isolated; the ratio of left ventricular weight to body weight and systolic blood pressure (SBP) were determined and heart morphometry was assessed after hematoxylin and eosin staining. Results indicated STS inhibited Ang II-induced increases in myocyte diameter and decreased the LVW/BW ratio independent of decreasing systolic blood pressure. In vitro, treatment of cultured cardiomyocytes with STS inhibited Ang II-induced increase in cell size, protein synthesis, ANP expression, activation of extracellular signalregulated kinase (ERK) and ERK kinase (MEK). Then we reexamined the mechanism of STS-induced antihypertrophic effects. Results revealed MEK inhibitor U0126 (20 µM) markedly enhanced STS-induced depressions in [3H]leucine incorporation and ANP expression. In conclusion, MEK/ERK pathway plays a significant role in the anti-hypertrophic effects of STS.
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