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Wei, Guoying,Jin, Yongdong,Ge, Hongliang,Luo, Jiuli Korean Chemical Society 2005 Bulletin of the Korean Chemical Society Vol.26 No.4
The Belousov-Zhabotinskii system measured by a Pt electrode is investigated under external electrode current constraint. A dynamical analysis of the electrode reaction phase has been made by means of a linearized stability criterion valid for three-variable system. It turns out that limit cycle oscillatory regime and dynamical behaviors of the electrode reaction phase have been degenerated under periodical electrode current.
Guoying Wei,Yongdong Jin,Hongliang Ge,Jiuli Luo 대한화학회 2005 Bulletin of the Korean Chemical Society Vol.26 No.4
The Belousov-Zhabotinskii system measured by a Pt electrode is investigated under external electrode current constraint. A dynamical analysis of the electrode reaction phase has been made by means of a linearized stability criterion valid for three-variable system. It turns out that limit cycle oscillatory regime and dynamical behaviors of the electrode reaction phase have been degenerated under periodical electrode current.
Pan Yibin,Yan Lili,Chen Qiaoqiao,Wei Cheng,Dai Yongdong,Tong Xiaomei,Zhu Haiyan,Lu Meifei,Zhang Yanling,Jin Xiaoying,Zhang Tai,Lin Xiaona,Zhou Feng,Zhang Songying 생화학분자생물학회 2021 Experimental and molecular medicine Vol.53 No.-
In early pregnancy, the placenta anchors the conceptus and supports embryonic development and survival. This study aimed to investigate the underlying functions of Shh signaling in recurrent miscarriage (RM), a serious disorder of pregnancy. In the present study, Shh and Gli2 were mainly observed in cytotrophoblasts (CTBs), Ptch was mainly observed in syncytiotrophoblasts (STBs), and Smo and Gli3 were expressed in both CTBs and STBs. Shh signaling was significantly impaired in human placenta tissue from recurrent miscarriage patients compared to that of gestational age-matched normal controls. VEGF-A and CD31 protein levels were also significantly decreased in recurrent miscarriage patients. Furthermore, inhibition of Shh signaling impaired the motility of JAR cells by regulating the expression of Gli2 and Gli3. Intriguingly, inhibition of Shh signaling also triggered autophagy and autolysosome accumulation. Additionally, knockdown of BECN1 reversed Gant61-induced motility inhibition. In conclusion, our results showed that dysfunction of Shh signaling activated autophagy to inhibit trophoblast motility, which suggests the Shh pathway and autophagy as potential targets for RM therapy.