RISS 학술연구정보서비스

검색
다국어 입력

http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.

변환된 중국어를 복사하여 사용하시면 됩니다.

예시)
  • 中文 을 입력하시려면 zhongwen을 입력하시고 space를누르시면됩니다.
  • 北京 을 입력하시려면 beijing을 입력하시고 space를 누르시면 됩니다.
닫기
    인기검색어 순위 펼치기

    RISS 인기검색어

      검색결과 좁혀 보기

      선택해제

      오늘 본 자료

      • 오늘 본 자료가 없습니다.
      더보기
      • 무료
      • 기관 내 무료
      • 유료
      • KCI등재

        Facile Synthesis of Nanosheet-Structured V2O5 with Enhanced Electrochemical Performance for High Energy Lithium-Ion Batteries

        Shuquan Liang,Mulan Qin,Yan Tang,Qing Zhang,Xilin Li,Anqiang Pan 대한금속·재료학회 2014 METALS AND MATERIALS International Vol.20 No.5

        Nanosheet-structured vanadium pentoxide (V2O5) has been fabricated by a sol-gel method. As revealed bythe TEM, the as-synthesized V2O5 crystallites are composed of layer-by-layer stacked nanosheets. As acathode material for lithium batteries, it exhibits much better electrochemical performance than the startingcommercial V2O5 powders. A high specific discharge capacity of 264 mA h g-1 can be obtained for thenanosheet-structured electrodes, which retains the capacity of 90% after 50 cycles. However, the commercialV2O5 only delivers a specific discharge capacity of 206 mA h g-1 with a capacity retention of 64%after 50 cycles. Moreover, the nanosheet-structured V2O5 electrodes show much-improved C-rate capability. The superior cycling performance demonstrates that the nanosheet-structured V2O5 is a promising cathodematerial in lithium-ion battery applications.

      • KCI등재

        Effect of Pass Deformation on Microstructure, Corrosion and Electrochemical Properties of Aluminum Alloy Anodes for Alkaline Aluminum Fuel Cell Applications

        Yong Zhang,Shuquan Liang,Athar Javed,Dikai Guan 대한금속·재료학회 2013 METALS AND MATERIALS International Vol.19 No.3

        The effect of pass deformation in rolling processes on the microstructure, corrosion resistance and electrochemical activity of Al-Mg-Bi-Sn-Ga-In alloy anodes operated in an alkaline solution (85 °C, 5 mol·L−1 NaOH with addition of NaSnO3) with current density of 800 mA·cm−2 was investigated. To analyze the microstructure of aluminum alloy anodes, we used scanning electron microscope, transmission electron microscope and electron backscatter diffraction techniques. We also used the hydrogen evolution method and electrochemical testing techniques to investigate the corrosion and electrochemical behavior of aluminum alloy anodes. The results showed that uniform microstructures with homogeneous distribution of fine active segregated phases as well as an excellent electrochemical performance of the aluminum alloy anodes were achieved by the dynamic recrystallization under pass deformation of 40%. We found that the aluminum alloy anodes (under pass deformation of 40%) had the lowest hydrogen evolution rate (0.092 mL·min−1·cm−2)and the most negative electrode potential (−1.585 V).

      • Suppression of PTRF Alleviates the Polymicrobial Sepsis Induced by Cecal Ligation and Puncture in Mice

        Zheng, Yijie,Lee, Seonjin,Liang, Xiaoliang,Wei, Shuquan,Moon, Hyung-Geun,Jin, Yang Oxford University Press 2013 The Journal of infectious diseases Vol.208 No.11

        <P><B><I>Background.</I></B> Sepsis and sepsis-associated organ failure are devastating conditions. Understanding the detailed cellular/molecular mechanisms involved in sepsis should lead to the identification of novel therapeutic targets.</P><P><B><I>Methods.</I></B> Cecal ligation and puncture (CLP) was used as a polymicrobial sepsis model in vivo to determine mortality and end-organ damage. Macrophages were adopted as the cellular model in vitro for mechanistic studies.</P><P><B><I>Results.</I></B> PTRF+/− mice survived longer and suffered less organ damage after CLP. Reductions in nitric oxide (NO) and iNOS biosynthesis were observed in plasma, macrophages, and vital organs in the PTRF+/− mice. Using an acute sepsis model after CLP, we found that iNOS−/− mice had a comparable level of survival as the PTRF+/− mice. Similarly, polymerase I transcript release factor (PTRF) deficiency resulted in decreased iNOS and NO/ROS production in macrophages in vitro. Mechanistically, lipopolysaccharide (LPS) enhanced the co-localization and interaction between PTRF and TLR4 in lipid rafts. Deletion of PTRF blocked formation of the TLR4/Myd88 complex after LPS. Consistent with this, lack of PTRF impaired the TLR4 signaling, as shown by the decreased p-JNK, p-ERK, and p-p38, which are upstream factors involved in iNOS transcription.</P><P><B><I>Conclusion.</I></B> PTRF is a crucial regulator of TLR4 signaling in the development of sepsis.</P>

      연관 검색어 추천

      이 검색어로 많이 본 자료

      활용도 높은 자료

      해외이동버튼