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Decomposition of gas-phase benzene using hybrid systems of non-thermal plasma and catalyst
Hyun-Ha Kim,Atsushi Ogata,Shigeru Futamura,Yong-Hwan Lee 한국물리학회 2004 THE JOURNAL OF THE KOREAN PHYSICAL SOCIETY Vol.44 No.52
This paper presents the decomposition of gas-phase benzene using a plasma-driven catalyst (PDC) reactor packed with Ag/TiO2. The amount of catalyst did not aect the decomposition of benzene or the selectivities of CO and CO2 in the PDC reactor. The decomposition of benzene was mostly determined by the specic input energy (SIE). The main gas-phase products of benzene decomposition were CO and CO2. At lower SIE, a trace amount of formic acid was detected as a minor product. The carbon balance, based on the sum of CO and CO2, also indicated that no other intermediates were formed in the decomposition of benzene using the PDC reactor. In comparison to the conventional pulsed corona discharge reactor and surface discharge reactor, nanometer-sized aerosol formation from the decomposition of benzene was negligible with the PDC reactor, which was also supported by the carbon balance data. The formation of N2O was slightly lower with a larger amount of Ag/TiO2 catalyst.
Kaneko, Takaaki,Shimpo, Kan,Chihara, Takeshi,Beppu, Hidehiko,Tomatsu, Akiko,Shinzato, Masanori,Yanagida, Takamasa,Ieike, Tsutomu,Sonoda, Shigeru,Futamura, Akihiko,Ito, Akihiro,Higashiguchi, Takashi Asian Pacific Journal of Cancer Prevention 2012 Asian Pacific journal of cancer prevention Vol.13 No.5
High temperature- and pressure-treated garlic (HTPG) has been shown to have enhanced antioxidative activity and polyphenol contents. Previously, we reported that HTPG inhibited 1,2-dimethylhydrazine-induced mucin depleted foci (premalignant lesions) and $O^6$-methylguanine DNA adduct formation in the rat colorectum. In the present study, we investigated the modifying effects of HTPG on N-ethyl-N'-nitro-N-nitrosoguanidine (ENNG)-induced pyloric stomach and small intestinal carcinogenesis in mice. Male C57BL/6 mice were given ENNG (100 mg/l) in drinking water for the first 4 weeks, then a basal diet or diet containing 2% or 5% HTPG for 30 weeks. The incidence and multiplicity of pyloric stomach and small intestinal (duodenal and jejunal) tumors in the 2% HTPG group (but not in the 5% HTPG group) were significantly lower than those in the control group. Cell proliferation of normal-appearing duodenal mucosa was assessed by MIB-5 immunohistochemistry and shown to be significantly lower with 2% HTPG (but again not 5% HTPG) than in controls. These results in dicate that HTPG, at 2% in the diet, inhibited ENNG-induced pyloric stomach and small intestinal (especially duodenal) tumorigenesis in mice, associated with suppression of cell proliferation.