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Negative gate bias and light illumination-induced hump in amorphous InGaZnO thin film transistor.
Jeon, Jae-Hong,Seo, Seung-Bum,Park, Han-Sung,Choe, Hee-Hwan,Seo, Jong-Hyun,Park, Kee-Chan,Park, Sang-Hee Ko American Scientific Publishers 2013 Journal of Nanoscience and Nanotechnology Vol.13 No.11
<P>While observing the transfer characteristics of a-IGZO TFTs, it was noticed that a hump occurred in the subthreshold regime after light and bias stress. This study analyzes the mechanism of the hump occurrence. It was determined that hump characteristics were related with parasitic TFTs which formed at the peripheral edges parallel with the channel direction. It seems that the negative shift of the transfer characteristics of parasitic TFTs was larger than that of the main TFT under light and bias stress. Therefore, the difference in the negative shift between the main TFT and the parasitic TFT was the origin of the hump occurrence. We investigated the instability of a-IGZO TFTs under negative gate bias with light illumination for various channel structures in order to verify the above mechanism.</P>
Positive feedback regulation of Akt‐FMRP pathway protects neurons from cell death
Jeon, Se Jin,Han, Seol‐,Heui,Yang, Sung‐,Il,Choi, Ji woong,Kwon, Kyoung Ja,Park, Seung Hwa,Kim, Hahn Young,Cheong, Jae Hoon,Ryu, Jong Hoon,Ko, Kwang Ho,Wells, David G,Shin, Chan Young Blackwell Publishing Ltd 2012 Journal of Neurochemistry Vol.123 No.2
<P><I>J. Neurochem.</I> (2012) <B>123</B>, 226–238.</P><P><B>Abstract</B></P><P>Fragile X syndrome (FXS), the most common single genetic cause of mental retardation and autistic spectrum disease, occurs when <I>FMR1</I> gene is mutated. <I>FMR1</I> encodes fragile X mental retardation protein (FMRP) which regulates translation of mRNAs playing important roles in the development of neurons as well as formation and maintenance of synapses. To examine whether FMRP regulates cell viability, we induced apoptosis in rat primary cortical neurons with glutamate <I>in vitro</I> and with middle cerebral artery occlusion (MCAO) in striatal neurons <I>in vivo</I>. Both conditions elicited a rapid, but transient FMRP expression in neurons. This up‐regulated FMRP expression was abolished by pre‐treatment with PI3K and Protein Kinase B (Akt) inhibitors: LY294002, Akt inhibitor IV, and VIII. Reduced FMRP expression <I>in vitro</I> or <I>in vivo</I> using small hairpin <I>Fmr1</I> virus exacerbated cell death by glutamate or MCAO, presumably <I>via</I> hypophosphorylation of Akt and reduced expression of B‐cell lymphoma‐extra large (Bcl‐xL). However, over‐expression of FMRP using enhanced green fluorescent protein (eGFP)‐FMRP constructs alleviated cell death, increased Akt activity, and enhanced Bcl‐xL production. The pro‐survival role of Akt‐dependent up‐regulation of FMRP in glutamate‐stimulated cultured neuron as well as in ischemic brain may have a clinical importance in FXS as well as in neurodegenerative disorders and traumatic brain injury.</P>
Cystoperitoneal Shunting after Fenestration of an Enlarging Arachnoid Cyst
Jeon, Ik-Chan,Kim, Min-Su,Kim, Seong-Ho,Jang, Sung-Ho 영남대학교 의과대학 2008 Yeungnam University Journal of Medicine Vol.25 No.2
A two-month-old girl with a history of an incidental arachnoid cyst in the prenatal period (38 weeks) presented with persistent irritability A follow-up computed tomographic (CT) scan revealed an enlarged arachnoid cyst with hydrocephalus We performed craniotomy and fenestration, but the cyst size did not decrease, and hydrocephalus had worsened on a follow-up CT scan performed 13 months after fenestration. The patient was treated with cystoperitoneal shunting Follow-up magnetic resonance imaging (MRI) performed 5 years later revealed that the arachnoid cyst had decreased in size and that the hydrocephalus had resolved. Enlarging arachnoid cysts are not common, and optimal surgical treatment is uncertain Based on the features of this case, we believe cystoperitoneal shunting is an advisable surgical intervention for patients with enlarging arachnoid cysts presenting with hydrocephalus.
Jeon, Nam Joong,Lee, Hag Geun,Kim, Young Chan,Seo, Jangwon,Noh, Jun Hong,Lee, Jaemin,Seok, Sang Il American Chemical Society 2014 JOURNAL OF THE AMERICAN CHEMICAL SOCIETY - Vol.136 No.22
<P>Three spiro-OMeTAD derivatives have been synthesized and characterized by <SUP>1</SUP>H/<SUP>13</SUP>C NMR spectroscopy and mass spectrometry. The optical and electronic properties of the derivatives were modified by changing the positions of the two methoxy substituents in each of the quadrants, as monitored by UV–vis spectroscopy and cyclic voltammetry measurements. The derivatives were employed as hole-transporting materials (HTMs), and their performances were compared for the fabrication of mesoporous TiO<SUB>2</SUB>/CH<SUB>3</SUB>NH<SUB>3</SUB>PbI<SUB>3</SUB>/HTM/Au solar cells. Surprisingly, the cell performance was dependent on the positions of the OMe substituents. The derivative with <I>o</I>-OMe substituents showed highly improved performance by exhibiting a short-circuit current density of 21.2 mA/cm<SUP>2</SUP>, an open-circuit voltage of 1.02 V, and a fill factor of 77.6% under 1 sun illumination (100 mW/cm<SUP>2</SUP>), which resulted in an overall power conversion efficiency (PCE) of 16.7%, compared to ∼15% for conventional <I>p</I>-OMe substituents. The PCE of 16.7% is the highest value reported to date for perovskite-based solar cells with spiro-OMeTAD. </P><P><B>Graphic Abstract</B> <IMG SRC='http://pubs.acs.org/appl/literatum/publisher/achs/journals/content/jacsat/2014/jacsat.2014.136.issue-22/ja502824c/production/images/medium/ja-2014-02824c_0004.gif'></P><P><A href='http://pubs.acs.org/doi/suppl/10.1021/ja502824c'>ACS Electronic Supporting Info</A></P>