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RISS 인기검색어
- 검색키워드
- 저자 : Qingde Zhang (검색결과 4 건)
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Qingde Zhang,Yisheng Tan,Caihong Yang,Hongjuan Xie 한국공업화학회 2013 Journal of Industrial and Engineering Chemistry Vol.19 No.3
The conversion of syngas to aromatics via dimethyl ether was investigated over MnCl2 modified HZSM-5zeolites. The results demonstrated that 2%MnCl2 modified HZSM-5 (SiO2/Al2O3 = 38) exhibited higher pxylene selectivity than other catalysts and further decreased 1,2,4,5-tetramethylbenzene selectivity. The CO conversion was obviously increased after 5%MnCl2 modification to HZSM-5. The catalysts were characterized by XRD, BET, XPS, FT-IR, NH3-TPD, SEM, element analysis and O2-TPO. The loading amount of MnCl2 affected the adsorption and reaction of DME molecules on zeolites. Appropriate amount of MnCl2 introduction could adjust the acidity and pore volume of HZSM-5 to increase p-xylene selectivity and CO conversion.
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Qingde Zhang,Yizhuo Han,Yisheng Tan,Guangbo Liu,Caihong Yang 한국공업화학회 2014 Journal of Industrial and Engineering Chemistry Vol.20 No.4
The promotional effects of Sm2O3 on Mn-H4SiW12O40/SiO2 for dimethyl ether (DME) direct-oxidation to dimethoxymethane (DMM) were investigated. The results showed that Sm2O3 introduction could significantly improve the activity of Mn-H4SiW12O40/SiO2 for DMM formation, and DMM selectivity was remarkably increased from 36.3% to 60.3% when the Sm2O3 content was 1%. The catalysts were characterized by ICP-AES, XRD, NH3-TPD, Pyridine-IR and XPS. The Sm2O3 introduction enhanced the number of Lewis acid sites and weak acid sites and also increased the amount of Mn4+ species of Mn-H4SiW12O40/SiO2, which is favorable for the formation of DMM.
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Role of Dehydrocorybulbine in Neuropathic Pain After Spinal Cord Injury Mediated by P2X4 Receptor
Zhongwei Wang,Wei Mei,Qingde Wang,Rundong Guo,Peilin Liu,Yuqiang Wang,Zijuan Zhang,Limin Wang 한국분자세포생물학회 2019 Molecules and cells Vol.42 No.2
Chronic neuropathic pain is one of the primary causes of disability subsequent to spinal cord injury. Patients experiencing neuropathic pain after spinal cord injury suffer from poor quality of life, so complementary therapy is seriously needed. Dehydrocorybulbine is an alkaloid extracted from Corydalis yanhusuo. It effectively alleviates neuropathic pain. In the present study, we explored the effect of dehydrocorybulbine on neuropathic pain after spinal cord injury and delineated its possible mechanism. Experiments were performed in rats to evaluate the contribution of dehydrocorybulbine to P2X4 signaling in the modulation of pain-related behaviors and the levels of pronociceptive interleukins and proteins after spinal cord injury. In a rat contusion injury model, we confirmed that chronic neuropathic pain is present on day 7 after spinal cord injury and P2X4R expression is exacerbated after spinal cord injury. We also found that administration of dehydrocorybulbine by tail vein injection relieved pain behaviors in rat contusion injury models without affecting motor functions. The elevation in the levels of pronociceptive interleukins (IL-1β, IL-18, MMP-9) after spinal cord injury was mitigated by dehydrocorybulbine. Dehydrocorybulbine significantly mitigated the upregulation of P2X4 receptor and reduced ATP-evoked intracellular Ca2+ concentration. Both P2XR and dopamine receptor2 agonists antagonized dehydrocorybulbine’s antinociceptive effects. In conclusion, we propose that dehydrocorybulbine produces antinociceptive effects in spinal cord injury models by inhibiting P2X4R.
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Role of Dehydrocorybulbine in Neuropathic Pain After Spinal Cord Injury Mediated by P2X4 Receptor
Wang, Zhongwei,Mei, Wei,Wang, Qingde,Guo, Rundong,Liu, Peilin,Wang, Yuqiang,Zhang, Zijuan,Wang, Limin Korean Society for Molecular and Cellular Biology 2019 Molecules and cells Vol.42 No.2
Chronic neuropathic pain is one of the primary causes of disability subsequent to spinal cord injury. Patients experiencing neuropathic pain after spinal cord injury suffer from poor quality of life, so complementary therapy is seriously needed. Dehydrocorybulbine is an alkaloid extracted from Corydalis yanhusuo. It effectively alleviates neuropathic pain. In the present study, we explored the effect of dehydrocorybulbine on neuropathic pain after spinal cord injury and delineated its possible mechanism. Experiments were performed in rats to evaluate the contribution of dehydrocorybulbine to P2X4 signaling in the modulation of pain-related behaviors and the levels of pronociceptive interleukins and proteins after spinal cord injury. In a rat contusion injury model, we confirmed that chronic neuropathic pain is present on day 7 after spinal cord injury and P2X4R expression is exacerbated after spinal cord injury. We also found that administration of dehydrocorybulbine by tail vein injection relieved pain behaviors in rat contusion injury models without affecting motor functions. The elevation in the levels of pronociceptive interleukins ($IL-1{\beta}$, IL-18, MMP-9) after spinal cord injury was mitigated by dehydrocorybulbine. Dehydrocorybulbine significantly mitigated the upregulation of P2X4 receptor and reduced ATP-evoked intracellular $Ca^{2+}$ concentration. Both P2XR and dopamine receptor2 agonists antagonized dehydrocorybulbine's antinociceptive effects. In conclusion, we propose that dehydrocorybulbine produces antinociceptive effects in spinal cord injury models by inhibiting P2X4R.
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