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Junrong Liu,James J. Sheng 한국공업화학회 2019 Journal of Industrial and Engineering Chemistry Vol.72 No.-
Surfactant enhanced spontaneous imbibition is an effective method to enhance oil recovery in shalereservoirs, however the mechanism is still unclear. This work attempts to investigate the effect of addedsurfactants on the spontaneous imbibition into shale rocks using NMR tests. Contact angles and IFT aremeasured to evaluate the efficiency of different surfactants in terms of wettability alteration and IFTreduction. According to the experimental results, the studied anionic surfactants perform better on thewettability alteration of shale rocks than studied nonionic surfactants, and the key factor to enhance theshale oil recovery may be wettability alteration.
Xiong Song,Liangui Nie,Junrong Long,Junxiong Zhao,Xing Liu,Liuyang Wang,Da Liu,Sen Wang,Shengquan Liu,Jun Yang 대한약리학회 2023 The Korean Journal of Physiology & Pharmacology Vol.27 No.1
Hypothyroidism alone can lead to myocardial fibrosis and result in heart failure, but traditional hormone replacement therapy does not improve the fibrotic situation. Hydrogen sulfide (H2S), a new gas signaling molecule, possesses antiinflammatory, antioxidant, and anti-fibrotic capabilities. Whether H2S could improve hypothyroidism-induced myocardial fibrosis are not yet studied. In our study, H2S could decrease collagen deposition in the myocardial tissue of rats caused by hypothyroidism. Furthermore, in hypothyroidism-induced rats, we found that H2S could enhance cystathionine-gamma-lyase (CSE), not cystathionine β-synthase (CBS), protein expressions. Finally, we noticed that H2S could elevate autophagy levels and inhibit the transforming growth factor-β1 (TGF-β1) signal transduction pathway. In conclusion, our experiments not only suggest that H2S could alleviate hypothyroidism- induced myocardial fibrosis by activating autophagy and suppressing TGF-β1/ SMAD family member 2 (Smad 2) signal transduction pathway, but also show that it can be used as a complementary treatment to conventional hormone therapy.