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Jianpeng Wu,Xumin Zhang,Kuan Jiang,Hongbing Jia,Xiaoting Rui,Fufeng Yang 한국섬유공학회 2020 Fibers and polymers Vol.21 No.8
In this work, a distinguished hydrothermal treatment succeeded in modifying aramid nanofibers (ANFs) with thehelp of mixed acid, and the hydrothermal-treated ANFs (hANFs) were regarded as a reinforcement agent to be introducedinto styrene-butadiene rubber (SBR) via latex co-agglutination treatment. The enhanced comprehensive behaviour of SBR/hANFs composites induced by π-π stacking interaction between hANFs and polymer chains were investigated in details. With respect to virgin SBR, it was found that the temperature at maximum rate of degradation, tear strength and tensilestrength of composites with adding 7 phr (parts per hundred rubber) hANFs significantly improved by 14 oC, 148 % and215 %, respectively. The incorporation of hANFs also endowed SBR with greatly enhanced solvent resistance.
Kaiyun Qin,Fenghua Zhang,Hongxia Wang,Na Wang,Hongbing Qiu,Xinzhuan Jia,Shan Gong,Zhengmao Zhang 생화학분자생물학회 2023 BMB Reports Vol.56 No.3
Ovarian cancer (OC) is the most common gynecological malignancyworldwide, and chemoresistance occurs in most patients,resulting in treatment failure. A better understanding of themolecular processes underlying drug resistance is crucial fordevelopment of efficient therapies to improve OC patient outcomes. Circular RNAs (circRNAs) and ferroptosis play crucialroles in tumorigenesis and resistance to chemotherapy. However,little is known about the role(s) of circRNAs in regulatingferroptosis in OC. To gain insights into cisplatin resistance inOC, we studied the ferroptosis-associated circRNA circSnx12. We evaluated circSnx12 expression in OC cell lines and tissuesthat were susceptible or resistant to cisplatin using quantitativereal-time PCR. We also conducted in vitro and in vivoassays examining the function and mechanism of lnc-LBCSs. Knockdown of circSnx12 rendered cisplatin-resistant OC cellsmore sensitive to cisplatin in vitro and in vivo by activatingferroptosis, which was at least partially abolished by downregulationof miR-194-5p. Molecular mechanics studies indicatethat circSnx12 can be a molecular sponge of miR-194-5p, whichtargets SLC7A11. According to our findings, circSnx12 amelioratescisplatin resistance by blocking ferroptosis via a miR-194-5p/SLC7A11 pathway. CircARNT2 may thus serve as an effectivetherapeutic target for overcoming cisplatin resistance in OC.