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南韓의 石炭의 反射度, X-線回折 및 安定同位元素 分析硏究
金奎漢,裵慶姬 梨花女子大學校 韓國生活科學硏究院 1988 韓國生活科學硏究院 論叢 Vol.41 No.-
Reflectance, X-ray diffraction pattern and isotope composition of eleven coal samples from seven major coal fields of Paleozoic to Mesozoic in age were studied to examine their mineral constituent, degree of coalification and graphitization. The relationships between chemical composition and degree of coalification and graphitization were also discussed. Under microscope, these coal samples were identified to be mostly collinite of vitrinite group with subordinate fusinite and semifusinite of inertinite group. Compositional ratios of vitrinite and inertinite in volume range from 94 : 6 to 67 : 33 indicating predominance of vitrinite over inertinite by two to 15 times. The bulk of coals comprise mainly macerals. Detrital minerals, however, contained 15 to 45 percent of the total volume of coal samples. The reflectances of vitrinite in most samples, except for a Boeun coal, are measured to be 3.5 to 4.0 Rm% indicating that these coal samples are equivalent to anthracite. This is also consistent of the data of X-ray diffraction patterns which better discriminate coal ranks than the reflectance does. Carbon isotopic compositions of coals and coaly shales from various coal fields range from -16.3 to -25.3%, with a mean value of -22.3%. No difference of carbon isotopic composition is found in various coals with different geologic age. It is apparent that the Najeon coal beds are getting higher in degree of graphitization with depth in the undeground. The reason for this can be attributed to a potential increase of hydrothermal alteration of the coal beds due to the quartz porphyry intrusion, by which an introduction of igneous quartz to the coal beds can be postulated to explain the gradual increase of ash contents of the coal.
김문령,김아미,배순희,연규월,전이리,조종남,한경덕,홍정애 이화여자대학교 의과대학 1972 梨花醫學誌 Vol.- No.5
With mice, a single intraperioneal injection of toxic dose of carbon tetrachloride is capable of inducing definite degeneration and necrosis of the hepatic calls of the central and midzonal regions of the lobules. The early changes are sinusoidal congestion, hydropic and fatty degeneration at six-twelve hours. In twenty-four hours, the cells of the central and midzonal region undergo complete necrosis which are soon invaded by inflammatory cells, leaving the mesenchymal supporting stroma and sinusoids unaffected. In three days, the necrotic cells in the central zone are disappearing and begin to be replaced by the newly formed hepatic cells. Repair is going on actively, chiefly through mitotic division of the healthy liver cells, not only at the borders of the area of necrosis but diffusely throughout the organ. The protal canals frequently invaded by inflammatory cells. By the seventh day, the dead tissue has removed. Repair is complete by the fourteenth day. There is no evidence of bile duct proliferation.