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최석호,이성훈,김학선,박종권 한밭대학교 정보통신전문대학원 2004 정보통신전문대학원 논문집 Vol.2 No.1
본 논문은 CPW급전 구조와 CST사의 Microwave Studio 사용하여 UWB 안테나를 설계하였다. 시물레이션 결과 제안된 안테나의 대역폭은 3.O5-13GHz를 가지고 방사패턴은 무지향성의 형태를 갖는 것을 보였다. In this paper, we have designed a UWB antenna using CPW feed line and used the Microwave Studio of CST to simulate the UWB antenna. Simulated results show that antenna bandwidth is 3.05-13GHz and radiation pattern is omni-directional.
서진욱,정재훈,최용의,이학성,신차균 중앙대학교 유전공학연구소 2003 遺傳工學硏究論集 Vol.16 No.1
The enzyme, squalene synthase, represents a branch point in the isoprenoid pathway capable of diverting carbon flow specifically to the biosynthesis of phytosterol and Triterpenoid. Transgenic Eleutherococcus senticosus plants were prepared by introducing the genes for squalene synthase derived from Panax ginseng (PgSS1), hygromycin phosphotransferase (HPT) and green fluorescent protein (GFP) through Agrobacterium-mediated transformation. Establishment of transgenic plantlets were confirmed by the presence of PgSS1 and HPT bands in the genomic DNA preparation using a PCR method and a Southern blotting. In addition, expression of the introduced GFP DNA was confirmed by observing green fluorescence of GFP from the embroyos. In the in vitro analysis of the PgSS1 enzymatic activities the transgenic plants showed to have 1.5 to 3 times higher than wild type plant, indicating that the PgSS1 genes was well over-expressed in the transgenic plants. Especially the PgSS1 acivities of embroyogenic cell was 120 times higher than those of plantlet.
전방 차량의 각도 변화에 따른 충돌방지장치 작동에 관한 연구
최형수(Hyeong-Su Choi),장우영(Woo-Young Jang),성수진(Su-Jin Seong),김종혁(Jong-Hyuk Kim),최지훈(Ji-Hun Choi),이학주(Hak-Ju Lee),최두현(Doo-Hyun Choi) 한국자동차공학회 2022 한국자동차공학회 학술대회 및 전시회 Vol.2022 No.11
ADAS, which can prevent accidents, is installed in many vehicles and is being actively developed and researched. In fact, a study combining data from five European countries and Australia found that low-speed AEB technology reduced rear-end collisions by 38%. In contrast, vehicle-to-vehicle side collisions cause the most injuries and deaths. In this paper, the relationship between the angle of the front vehicle and the operation of the collision avoidance system (AEB, FCW) is investigated through an actual vehicle test. To figure out limit angle of the collision avoidance system, the test is performed on three vehicles by changing the angle of the forward vehicle.
Choi, Yong-Keun,Kumaran, Rangarajulu Senthil,Jeon, Hyeon Jin,Song, Hak-Jin,Yang, Yung-Hun,Lee, Sang Hyun,Song, Kyung-Guen,Kim, Kwang Jin,Singh, Vijay,Kim, Hyung Joo Elsevier 2015 Spectrochimica acta. Part A, Molecular and biomole Vol.145 No.-
<P><B>Abstract</B></P> <P>Microbial algal system can serve as a potential source for the production of much high value bioproducts and biofuels. The quality and intensity of light are the key elements to optimize the production of algal biomass and fatty acid contents. This study presents the effect of differential LED flashing light conditions on the growth of microalgae, <I>Acutodesmus obliquus</I>. The induced light stress was optimized for its biomass and fatty acid content. The microalgae are exposed to various frequency of intermittent LED flashing light (blue and red lights) at three different phases in the 18day cell growth (log, lag and stationary phase). The frequency of light flashing rate was adjusted to 120, 10, 5, 3.75, and 1times per min. The effect of light stress on growth and fatty acids composition of <I>A. obliquus</I> induced an increase in algae growth and fatty acid production. Different optimal timing for light stress was subjected to elucidate the effect of light stress on algae growth and fatty acid production. The results showed an increase in the algae growth (1.2mg/L of chl a content) under light stress condition at FT10 (flashing time, 10times per min) from the initial day (log phase) compared with the control experiment (0.4mg/L of chl a content). However, the total fatty acids (71mg/g) and volumetric FAME production (9.4ml/l) level was found to be significant under FT5 (flashing time, 5times per min), adopting flashing light from day 10 (stationary phase). TEM studies also revealed the deposition of lipid to be largest in the 18day old cells under flashing light (FT5) condition, representing maximum accumulation of lipids bodies (up to 770nm diameter in particle size) occupying approximately 42% of the total area of the cell.</P> <P><B>Highlights</B></P> <P> <UL> <LI> LED light stress on algal biomass and fatty acid production were studied. </LI> <LI> Flashing lights exposed on log, lag, and stationary phases. </LI> <LI> FT10 showed an increase in the algae growth. </LI> <LI> Total fatty acids and volumetric FAME production were higher under FT5. </LI> <LI> Deposition of lipid was found maximum in the 18day old cells (FT5). </LI> </UL> </P> <P><B>Graphical abstract</B></P> <P>[DISPLAY OMISSION]</P>
Carnitine induces autophagy and restores high-fat diet-induced mitochondrial dysfunction
Choi, Jin Woo,Ohn, Jung Hun,Jung, Hye Seung,Park, Young Joo,Jang, Hak Chul,Chung, Sung Soo,Park, Kyong Soo W.B. Saunders Co. [etc.] 2018 Metabolism, clinical and experimental Vol.78 No.-
<P><B>Abstract</B></P> <P><B>Objective</B></P> <P>Autophagy is suppressed in skeletal muscle and the liver with insulin resistance induced by a high-fat diet. Autophagy is essential for maintaining mitochondrial function, and dysfunctional mitochondria are associated with insulin resistance. As carnitine treatment is well known to improve insulin resistance by promoting mitochondrial function, we investigated if carnitine affects autophagy in the skeletal muscle of a high-fat diet-induced rodent model of obesity.</P> <P><B>Results</B></P> <P>After 6weeks on a high-fat diet (48kcal% fat), mice developed glucose intolerance, and the gastrocnemius muscle showed a decrease in insulin signaling and mitochondrial function, which was reversed after carnitine (100mg/kg/day) treatment by oral gavage for 2weeks. Swollen mitochondria with destroyed cristae were observed in the skeletal muscle of high-fat diet-fed mice but were not there after carnitine treatment. High-fat diet decreased LC3B-II, a marker of autophagosome formation, and increased sequestosome 1 (SQSTM1), expression of which was reversed after carnitine treatment. In C2C12 myotubes, prolonged treatment with palmitate suppressed autophagy, which was relieved by carnitine treatment. However, the induction of autophagy by carnitine in C2C12 myotubes was not observed after knock-down of peroxisome proliferator-activated receptor γ (PPARγ), which is known to regulate autophagy.</P> <P><B>Conclusion</B></P> <P>We conclude that the removal of dysfunctional mitochondria by induction of autophagy through PPARγ may be a novel mechanism by which carnitine improves insulin resistance and mitochondrial dysfunction in obesity.</P>