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Hongli Yu,Hao Wu,Yaozong Pan,Xiuyun Ge,Qian Zhang,Fagen Zhu,Baochang Cai 대한약학회 2015 Archives of Pharmacal Research Vol.38 No.10
The present study aimed at investigating thealum-processing mechanism attenuating toxicity of AraceaePinellia ternata and Pinellia pedatisecta. Animalretroperitoneal inflammatory model in vivo andmacrophagocyte release inflammatory factor model in vitrowere used to detect the effect of alum processing onraphides and lectin. Scanning electron microscopy wasused to observe the change in raphides during processing;HPLC method was used to determine the correlation betweenthe dissolution and corrosion of raphides and ion inthe alum solution; 27Al-NMR technology was used to detectthe relationship between aluminum oxalate complexformation and the dissolved and corrosion of raphides. Thechange in protein peptide sequence of lectin during theprocessing of alum solution was determined by ShotgunLC–MS assay. Raphides induced severe rabbit conjunctivaledema and an intraperitoneal injection of lectin increasedPGE2 and protein in mice peritoneal exudate, whiledecreased after treatment with alum solution processing. During the processing raphides was dissolved and corroded,then its structure was damaged. Raphides was soaked inthe alum solution and significantly decreased the oxalatecontent, and the effect was related with Al3? in the alum. Al3? in the alum combined with C2O42- of raphides into astable complex compound promoted the dissolution ofcalcium oxalate. Raphides soaked in the alum made lectinproteins dissolve, whereas protein peptide sequence oflectin was changed and the protein structure was damaged. Conclusion: Alum solution could decrease the toxicity ofP. ternata (Thunb.) Breit. and P. pedatisecta Schott. Sinceit made a special crystal structure of raphides damage andthe protein of lectin dissolve. The structure of toxic substancessignificantly changed, which decreased the inflammatoryeffect.
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Family History of Cancer and Head and Neck Cancer Risk in a Chinese Population
Huang, Yu-Hui Jenny,Lee, Yuan-Chin Amy,Li, Qian,Chen, Chien-Jen,Hsu, Wan-Lun,Lou, Pen-Jen,Zhu, Cairong,Pan, Jian,Shen, Hongbing,Ma, Hongxia,Cai, Lin,He, Baochang,Wang, Yu,Zhou, Xiaoyan,Ji, Qinghai,Zho Asian Pacific Journal of Cancer Prevention 2015 Asian Pacific journal of cancer prevention Vol.16 No.17
Background: The aim of this study was to investigate whether family history of cancer is associated with head and neck cancer risk in a Chinese population. Materials and Methods: This case-control study included 921 cases and 806 controls. Recruitment was from December 2010 to January 2015 in eight centers in East Asia. Controls were matched to cases with reference to sex, 5-year age group, ethnicity, and residence area at each of the centers. Results: We observed an increased risk of head and neck cancer due to first degree family history of head and neck cancer, but after adjustment for tobacco smoking, alcohol drinking and betel quid chewing the association was no longer apparent. The adjusted OR were 1.10 (95% CI=0.80-1.50) for family history of tobacco-related cancer and 0.96 (95%CI=0.75-1.24) for family history of any cancer with adjustment for tobacco, betel quid and alcohol habits. The ORs for having a first-degree relative with HNC were higher in all tobacco/alcohol subgroups. Conclusions: We did not observe a strong association between family history of head and neck cancer and head and neck cancer risk after taking into account lifestyle factors. Our study suggests that an increased risk due to family history of head and neck cancer may be due to shared risk factors. Further studies may be needed to assess the lifestyle factors of the relatives.
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