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DISCOVERY OF AN UNIDENTIFIED <i>FERMI</i> OBJECT AS A BLACK WIDOW-LIKE MILLISECOND PULSAR
Kong, A. K. H.,Huang, R. H. H.,Cheng, K. S.,Takata, J.,Yatsu, Y.,Cheung, C. C.,Donato, D.,Lin, L. C. C.,Kataoka, J.,Takahashi, Y.,Maeda, K.,Hui, C. Y.,Tam, P. H. T. IOP Publishing 2012 ASTROPHYSICAL JOURNAL LETTERS - Vol.747 No.1
<P>The Fermi gamma-ray Space Telescope has revolutionized our knowledge of the gamma-ray pulsar population, leading to the discovery of almost 100 gamma-ray pulsars and dozens of gamma-ray millisecond pulsars (MSPs). Although the outer-gap model predicts different sites of emission for the radio and gamma-ray pulsars, until now all of the known gamma-ray MSPs have been visible in the radio. Here we report the discovery of a 'radio-quiet' gamma-ray-emitting MSP candidate by using Fermi, Chandra, Swift, and optical observations. The X-ray and gamma-ray properties of the source are consistent with known gamma-ray pulsars. We also found a 4.63 hr orbital period in optical and X-ray data. We suggest that the source is a black widow-like MSP with a similar to 0.1 M-circle dot late-type companion star. Based on the profile of the optical and X-ray light curves, the companion star is believed to be heated by the pulsar while the X-ray emissions originate from pulsar magnetosphere and/or from intrabinary shock. No radio detection of the source has been reported yet, and although no gamma-ray/radio pulsation has been found we estimate that the spin period of the MSP is similar to 3-5 ms based on the inferred gamma-ray luminosity.</P>
Santangelo, S.,Messina, G.,Faggio, G.,Willinger, M.G.,Pinna, N.,Donato, A.,Arena, A.,Donato, N.,Neri, G. Elsevier 2010 Diamond and related materials Vol.19 No.5
Commercial tubular carbon nanofibers were uniformly coated with a 5nm thick vanadium oxide layer via a modified approach to atomic layer deposition. The composition and microstructure of the resulting hybrid material was analyzed by micro-Raman spectroscopy. The effect of the post-synthesis thermal treatment in air at temperatures in the range of 25-375<SUP>o</SUP>C was investigated in order to more deeply understand the behavior of the hybrid material in gas sensing devices. The obtained results demonstrate that the thermal treatment primarily affects the oxide coating-layer that is responsible for the sensing properties. The best sensor performance was obtained at the temperature at which the oxide layered-structure exhibits the highest structural order.
Oxygen-Sensing Properties of Electrospun CNTs/PVAc/TiO2 Composites
P. Frontera,S. Trocino,A. Donato,P.L. Antonucci,M. Lo Faro,G. Squadrito,G. Neri 대한금속·재료학회 2014 ELECTRONIC MATERIALS LETTERS Vol.10 No.1
Multi-walled-carbon-nanotubes/polyvinylacetate/titanium oxide (MWCNTs/PVAc/TiO2) composite fibers with different MWCNT loadings were prepared using the electrospinning technique. The addition of cetyl trimethyl ammonium bromide (CTAB) as a surfactant agent to promote the dispersion of the MWCNTs was also evaluated. The morphological and microstructural properties of as-spun and annealed samples in air at 600°C were examined using thermogravimetric analysis (TGA), Fourier transform infrared spectroscopy (FT-IR), scanning electron microscopy (SEM) and x-ray diffraction (XRD). The sensing characteristics of the synthesized TiO2-based nanostructures were evaluated for oxygen monitoring at moderate temperatures. The effects of the MWCNT loading, the addition of the surfactant and the composition of the TiO2 crystalline phases were investigated and discussed.
Inflammation promotes synucleinopathy propagation
Kim Tae-Kyung,Bae Eun-Jin,Jung Byung Chul,Choi Minsun,Shin Soo Jean,Park Sung Jun,Kim Jeong Tae,Jung Min Kyo,Ulusoy Ayse,Song Mi-Young,Lee Jun Sung,Lee He-Jin,Di Monte Donato A.,Lee Seung-Jae 생화학분자생물학회 2022 Experimental and molecular medicine Vol.54 No.-
The clinical progression of neurodegenerative diseases correlates with the spread of proteinopathy in the brain. The current understanding of the mechanism of proteinopathy spread is far from complete. Here, we propose that inflammation is fundamental to proteinopathy spread. A sequence variant of α-synuclein (V40G) was much less capable of fibril formation than wild-type α-synuclein (WT-syn) and, when mixed with WT-syn, interfered with its fibrillation. However, when V40G was injected intracerebrally into mice, it induced aggregate spreading even more effectively than WT-syn. Aggregate spreading was preceded by sustained microgliosis and inflammatory responses, which were more robust with V40G than with WT-syn. Oral administration of an anti-inflammatory agent suppressed aggregate spreading, inflammation, and behavioral deficits in mice. Furthermore, exposure of cells to inflammatory cytokines increased the cell-to-cell propagation of α-synuclein. These results suggest that the inflammatory microenvironment is the major driver of the spread of synucleinopathy in the brain.