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이상철 ( Sang Chul Lee ),손영웅 ( Young Woong Son ),심다운 ( Da Woon Sim ),박경희 ( Kyung Hee Park ),이재현 ( Jae-hyun Lee ),박중원 ( Jung-won Park ) 대한천식알레르기학회(구 대한알레르기학회) 2016 Allergy Asthma & Respiratory Disease Vol.4 No.6
Oral allergy syndrome (OAS) is caused by cross-reactivity between certain pollens and plant foods, including vegetables, nuts, or fruits. Here, we experienced 2 cases of OAS patients associated with mugwort pollinosis without sensitization to Fagales. A 54-year-old female repeatedly experienced skin rashes, perioral edema, nasal obstruction after eating fresh vegetables (celery, lettuce, chicory, radish sprouts, ginseng, etc.). She had suffered from allergic rhinitis worsening in autumn for 5 years. Specific IgE (sIgE) titers to ragweed and mugwort were elevated to 54.1 and 24.9 kU/L, respectively. With regard to the allergen component of pollens, sIgE to Art v 1 (mugwort) and Amb a 1 (ragweed) were elevated to 21.9 and 36.1 kU/L, respectively. Birch sIgE (including Bet v 1 and Bet v 2) was not detected. A 35-year-old male suffered from abdominal pain, skin rashes after eating mango and kiwi. In addition, systemic allergic reaction developed after consumption of tomato and ginseng. He had chronic rhinitis. The sIgE levels to ragweed, mugwort, and tomato were elevated to 0.55, 6.39, and 0.78 kU/L, respectively. The sIgE test results were all negative for Amb a 1, Bet v 1, and Bet v 2 sIgE. Specific IgE levels to Art v 1, Art v 2 sIgE were 3.51 and 4.46 kU/L, respectively. Based on the history and sIgE test results, 2 cases OAS were related to mugwort. We experienced 2 cases of weed pollinosis related to OAS. Culprit foods of OAS can vary depending on their cuisine cultures. (Allergy Asthma Respir Dis 2016:4:458-461)
가성낭을 동반한 만성 췌장염 환자에서 발생한 비장경색 및 비장파열 1 예
조용구(Yong Gu Cho),임영찬(Young Chan Lim),한병호(Beoung Ho Han),김충기(Chung Kee Kim),임홍섭(Hong Seub Lim),심영웅(Young Woong Sim),송갑영(Gap Young Song) 대한소화기학회 2001 대한소화기학회지 Vol.38 No.6
Splenic infarction is not common disease which is caused by occlusion of the main splenic artery or any of its branches. It is mainly caused by emboli that arise from cardiovascular disease, but sometimes by regional thrombosis associated with leukemia, myeloproliferative disorder, sickle cell anemia, pancreatitis, portal hypertension, splenic torsion, septicemia and drugs, etc. Splenic infarction accompained by chronic pancreatitis is most likely due to splenic vein thrombosis, surrounding tissue fibrosis accompanied with splenic vessel occlusion, and intrasplenic penetration of pancreatic pseudocyst. Recently, we experienced a case of splenic infarction and rupture in a 46-year-old male who had been diagnosed as chronic pancreatitis with pseudocyst. (Korean J Gastroenterol 2001;38:457-460)