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        혈액투석중 발생한 급성 고칼슘혈증

        이용준(Yong Joon Lee),김종진(Chong Jin Kim),김성경(Sung Kyung Kim),이민상(Min Sang Lee),홍관수(Kwan Su Hong),구완서(Whan Suh Koo),장윤식(Yoon Sik Chang),방병기(Byung Kee Bang) 대한내과학회 1987 대한내과학회지 Vol.32 No.5

        N/A We experienced two episodes of acute hypercalcemia during hemodialysis. In all case of 16 patients who were supplied with water by the central water reservoir system developed nausea, vomiting, abdominal pain, perioral paresthesia, and high blood pressure at 3 hours after initiation of hemodialysis, but one patient who used single water reservoir system did not develope any symptoms. The serum and dialysate calcium concentration measured when the symptoms developed rose to 12.3±1.6 mg/dl and 13.7±2.4 mg/dl at first episode, 13.6±1.0 mg/dl and 16.1 mg/dl at second episode, but the raw water calcium conecntration was 1.5 mg/dl and l. 4 mg/ dl. So it was considered that above symptoms were results from hypercalcemia which induced by abrupt release of calcium plug attached to the calcium exchange resin into water supply system. They were treated with further hemodialysis using raw water mixed dialysate without water softner. Thereafter serum calcium concentration decreased to 11.6±1.1 mg/ dl for earlier 3 hours of prolonged hemodialysis without softner, but 6 hours later serum calcium concentration rather increased because of volume depletion effects. We also evaluated the changes of serum calcium concentration after hemodialysis by using high calcium concentration concentrate and low calcium concentration concentrate. The serum calcium concentration after hemodialysis by using high calcium concentration concentrate significantly increased to 12.1±1.0 mg/dl as compared with 10.8±1.1mg/dl in using low calcium concentration concentrate hemodialysis.

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