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Interface damage mechanism of rubber cord composite material
Xiaohui Guo,Xiaojing Yuan,Guangyong Liu,Hefang Qiu,Fanfan Cui 한양대학교 청정에너지연구소 2023 Journal of Ceramic Processing Research Vol.24 No.1
In order to study the interface failure mechanism of rubber cord composites laid at symmetrical angles, a representativevolume element was established by the finite element method to simulate the stress distribution at the mesoscopic level ofrubber cord composites. The energy-based power law is used to simulate the damage evolution of the cord-rubber interface. The results show that the interface damage is caused by the torque that the cord bears during the stretching process, and theinterface assumes the role of transmitting torque in this process, so the failure occurred.The initial interface stiffness is a vitalbonding interface parameter that affects the interface damage evolution, and the initial stiffness is positively correlated withthe interface damage evolution variable. The increase in the included angle of the cord will cause the torque on the cord toincrease, resulting in aggravated interface failure.
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Effect of Chito-oligosaccharide Supplementation on Immunity in Broiler Chickens
Deng, Xingzhao,Li, Xiaojing,Liu, Pai,Yuan, Shulin,Zang, Jianjun,Li, Songyu,Piao, Xiangshu Asian Australasian Association of Animal Productio 2008 Animal Bioscience Vol.21 No.11
This study was conducted to determine the effects of dietary supplementation of either 100 mg/kg chito-oligosaccharide (COS) or chlortetracycline (CTC) with corn-soybean-fish meal on immunity in broiler chickens. A total of 147 one-day old male broiler chicks were randomly allocated to 3 treatments with 7 replicate pens per treatment and 7 birds per pen. The experimental diets consisted of a control diet based on corn, soybean and fish meal without COS and any antibiotic supplement and similar diets supplemented with either CTC (80 mg/kg from d 1 to 21 and 50 mg/kg from d 22 to 42) or COS (100 mg/kg from d 1 to 42). During the entire experimental period, all birds had ad libitum access to diets and water. The main immune organ indices, T-lymphocyte proliferation, serum cytokine concentrations, serum NO level and serum iNOS activity were measured on d 21 and d 42. On d 21, broilers fed 100 mg/kg COS had improved (p<0.01) indices of spleen, thymus, and bursa of Fabricius compared with the control and CTC birds. Birds receiving 100 mg/kg COS had higher (p<0.05) serum concentrations of $IL-1{\beta}$, IL-6, IgM, NO and iNOS than birds on the control treatment. Serum $Ca^{2+}$ level of birds fed 100 mg/kg COS tended to be higher (p = 0.049) than in birds fed CTC. On d 42, the birds fed 100 mg/kg COS had higher (p<0.05) concentrations of TNF-${\alpha}$ and IgM in serum than birds in both the CTC and control treatments. Birds fed 100 mg/kg COS had a higher concentration of IFN-$\gamma$ than the control group. In conclusion, dietary supplementation of COS appeared to improve the immunity of broilers by promoting the weight of the main immune organs, increasing IgM secretion, stimulating microphages to release $TNF-{\alpha}$, $IL-1{\beta}$, IL-6 and IFN-$\gamma$, and activating iNOS to induce NO.
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Yongpeng Ma,Dongli Yuan,Xiaojing Zhang,Zan Qu,Wenjun Huang 대한환경공학회 2021 Environmental Engineering Research Vol.26 No.2
In this work, we investigated the absorption characteristics of SO₂ and the effect of inhibitors on the desulfurization performances of Na₂SO₃. The results showed that the NO₂ had a competitive effect with SO₂ on SO₃<SUP>2-</SUP> which resulted in a significant decrease in the absorption capacity of SO₂. O₂ in the flue gas could decrease the absorption capacity of SO₂ due to the oxidation of Na₂SO₃. Besides, Na₂S₂O₃ had more excellent inhibiting effect on the oxidation of SO₃<SUP>2-</SUP>; the inhibition mechanism is understood on the basis of the free radical chain reaction, whereby S₂O₃<SUP>2-</SUP> combined with the sulfite free radical to form an inert substance, thus, quenching the reaction of free radical with the dissolved oxygen and invariably inhibiting the oxidation of SO₃<SUP>2-</SUP>. Furthermore, the intrinsic and the apparent oxidation kinetics of Na₂SO₃ oxidation process with Na₂S₂O₃ were investigated to explain the relationships between consumption rates of SO₃<SUP>2-</SUP> and the absorption capacities of SO₂ under different components in flue gas and absorption solution.
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HCBP6 upregulates human SREBP1c expression by binding to C/EBPβ-binding site in the SREBP1c promoter
( Xueliang Yang ),( Ming Han ),( Shunai Liu ),( Xiaoxue Yuan ),( Xiaojing Liu ),( Shenghu Feng ),( Li Zhou ),( Yaru Li ),( Hongping Lu ),( Jun Cheng ),( Shumei Lin ) 생화학분자생물학회(구 한국생화학분자생물학회) 2018 BMB Reports Vol.51 No.1
Sterol regulatory element-binding protein-1c (SREBP1c) plays an important role in triglyceride (TG) homeostasis. Although our previous study showed that hepatitis C virus core-binding protein 6 (HCBP6) regulates SREBP1c expression to maintain intracellular TG homeostasis, the mechanism underlying this regulation is unclear. In the present study, we found that HCBP6 increased intracellular TG levels by upregulating SREBP1c expression. HCBP6 increased SREBP1c transcription by directly binding to the SREBP1c promoter (at the -139- to +359-bp region). Moreover, we observed that HCBP6 interacted with C/EBPβ-binding site in the SREBP1c promoter both in vitro and in vivo. These results indicate that HCBP6 upregulates human SREBP1c expression by binding to the C/EBPβ-binding site in the SREBP1c promoter. [BMB Reports 2018; 51(1): 33-38]
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