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Choi, Sung Min,Lee, Jong-Heun,Choi, Moon-Bong,Hong, Jongsup,Yoon, Kyung Joong,Kim, Byung-Kook,Lee, Hae-Weon,Lee, Jong-Ho The Electrochemical Society 2015 Journal of the Electrochemical Society Vol.162 No.7
<P>The total electrical conductivity of BaCeO<SUB>3</SUB>-based proton conductors with various dopants was measured using a DC-4 probe method. Yb and In were used as dopants to increase electrical conductivity and chemical stability, respectively. The electronic and ionic partial conductivities of samples of various compositions, i.e., BaCe<SUB>1-</SUB><I><SUB>x</SUB></I><SUB>-</SUB><I><SUB>y</SUB></I>Yb<I><SUB>x</SUB></I>In<I><SUB>y</SUB></I>O<SUB>3-δ</SUB>, where <I>x</I> = 0.1, <I>y</I> = 0.00, 0.05, and 0.10 (BC10Yb, BC10Yb05In, and BC10Yb10In, respectively) and <I>x</I> = 0.15, <I>y</I> = 0.00 (BC15Yb), were calculated based on the relevant defect chemistry model and interpreted as functions of fairly extensive ranges of <I>P</I>(O<SUB>2</SUB>) (−4 ≤ log <I>P</I>(O<SUB>2</SUB>) ≤ 0) and <I>P</I>(H<SUB>2</SUB>O) (−3.5 ≤ log <I>P</I>(H<SUB>2</SUB>O) ≤ −1.5) at 700°C. The partial conductivities of all charge carriers, i.e., protons, holes, and oxygen vacancies, increased with Yb doping but decreased as In doping increased. The variations in the partial conductivity of holes and protons induced by the composition of doping constituents can be explained by the difference in electronegativity between dopant cations and oxygen anions as well as A- and B-site cations that are most critical in a general hopping conduction mechanism. Oxygen ion conductivity is controlled by the dopant size and content, which generally alter the lattice spacing and distortion of the perovskite structure.</P>
Sang Nag Ahn(安相洛),Kook Hyun Chung(鄭國鉉),Ha Cheol Hong(洪夏鐵),Hae Chune Choi(崔海椿),Huhn Pal Moon(文憲八) 한국육종학회 1996 한국육종학회지 Vol.28 No.1
This study was carried out to elucidate the inheritance of aroma in rice. Aromatic nature was tested with leaves of two aromatic/non-aromatic crosses and six crosses among aromatic cultivars. All F₁ hybrids of non-aromatic cultivar Hwaseongbyeo and aromatic cultivars, Sanghaehyanghyeolla, Miyakaori, Della, Selatus Malam, Manong Balay, Hyangmibyeo 1 and Chokoto were not aromatic and this seemed to indicate the recessive nature of the aroma in all tested rices. F₂ segregation ratio of 3 non-aromatic : 1 aromatic individual suggested that Sanghaehyanghyeolla and Miyakaori each contained a single gene for aroma. The finding that all F₁ hybrids among six aromatic rice cultivars were aromatic suggested that the aroma in Sanghaehyanghyeolla and Miyakaori may be controlled by the same gene known to be in Della and the aroma genes in Manong Balay and Chokoto are also allelic although the number of genes controlling the aroma in these cultivars are not known. No linkage was detected between the aroma gene(fgr) in Sanghaehyanghyeolla and two genetic markers(sug and v-8) located on rice chromosome 8.
Opuntia humifusa Ameliorated Cerulein-Induced Acute Pancreatitis
Choi, Sun Bok,Bae, Gi-Sang,Park, Kyoung-Chel,Jo, Il-Joo,Seo, Seung-Hee,Song, Kyung,Lee, Dong-Sung,Oh, Hyuncheol,Kim, Youn-Chul,Kim, Jong-Jin,Shin, Yong Kook,Park, Jin-Han,Seo, Min-Jun,Song, Ho-Joon,Pa by Lippincott Williams Wilkins. 2014 PANCREAS Vol.43 No.1
OBJECTIVE: The aim of this study was to evaluate the effects of Opuntia humifusa (OH) on cerulein-induced acute pancreatitis (AP). METHODS: Acute pancreatitis was induced via intraperitoneal injection of cholecystokinin analog cerulein (50 μg/kg). In the OH pretreatment group, OH was administered intraperitoneally (100, 250, or 500 mg/kg) 1 hour before first cerulein injection. In the posttreatment group, OH was administered intraperitoneally (500 mg/kg) 1 hour after the first cerulein injection. Furthermore, we isolated the pancreatic acinar cells using collagenase method, then investigated the acinar cell viability, cytokine productions, and the regulating mechanisms. RESULTS: The both pretreatment and posttreatment of OH treatment attenuated the severity of AP, as shown by the histology of the pancreas and lung, and inhibited neutrophil infiltration; serum amylase and lipase activities; proinflammatory cytokine expression such as interleukin 1, interleukin 6, and tumor necrosis factor α; and cell death including apoptosis and necrosis. Furthermore, OH inhibited the activation of c-Jun N-terminal kinases. CONCLUSIONS: These results suggest that OH reduces the severity of AP by inhibiting acinar cell death through c-Jun N-terminal kinases.