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- 저자 : Jinxia Wang (검색결과 3 건)
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Yang Yang,Jinxia Wang,Qingsong Duan,Chao Su,Mingjiao Yan,Yijia Dong 대한토목학회 2018 KSCE JOURNAL OF CIVIL ENGINEERING Vol.22 No.12
This paper investigated and simulated the mechanical effect of root reinforcing soil and stabilizing slope. The unconfined compression test was used to investigate the reinforcement soil behavior of herb roots. This paper used the 3D finite element to quantify the mechanical effect of the herb root for slope’s stability in terms of the safety factor. The soil was simulated by an elasticplastic model, and the root was simulated by a linear elastic model. Results show that using the Root Content (RC) to estimate the additional cohesion cr of Setaria sphacelata and Orchardgrass was a simple and reliable method. Lateral fine roots can play a more significant role in reinforcing soils and slopes. Compared to various slope angles, the steep slope was less stable. The slope existed a slope angle threshold αthr. When the slope angle α was smaller than αthr, the herb root system can improve the stability of slope. The α existed an optimal angle αopt for the root system. When α = αopt and α < αthr, the herb roots can exert its maximum stabilization capacity. Moreover, we can select herb species with lateral fine roots and more roots in deeper soil layer. Results can better estimate the cr for the herb of lateral fine roots and select the herb and the slope’s geometry in the engineering.
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Deep Learning-Based Modulation Detection for NOMA Systems
( Wenwu Xie ),( Jian Xiao ),( Jinxia Yang ),( Ji Wang ),( Xin Peng ),( Chao Yu ),( Peng Zhu ) 한국인터넷정보학회 2021 KSII Transactions on Internet and Information Syst Vol.15 No.2
Since the signal with strong power need be demodulated first for successive interference cancellation (SIC) receiver in non-orthogonal multiple access (NOMA) systems, the base station (BS) need inform the near user terminal (UT), which has allocated higher power, of the far UT’s modulation mode. To avoid unnecessary signaling overhead of control channel, a blind detection algorithm of NOMA signal modulation mode is designed in this paper. Taking the joint constellation density diagrams of NOMA signal as the detection features, the deep residual network is built for classification, so as to detect the modulation mode of NOMA signal. In view of the fact that the joint constellation diagrams are easily polluted by high intensity noise and lose their real distribution pattern, the wavelet denoising method is adopted to improve the quality of constellations. The simulation results represent that the proposed algorithm can achieve satisfactory detection accuracy in NOMA systems. In addition, the factors affecting the recognition performance are also verified and analyzed.
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TRIM56 Suppresses Multiple Myeloma Progression by Activating TLR3/TRIF Signaling
Ying Chen,Jing Zhao,Dengzhe Li,Jinxia Hao,Pengcheng He,Huaiyu Wang,Mei Zhang 연세대학교의과대학 2018 Yonsei medical journal Vol.59 No.1
Purpose: Tripartite-motif-containing protein 56 (TRIM56) has been found to exhibit a broad antiviral activity, depending upon E3 ligaseactivity. Here, we attempted to evaluate the function of TRIM56 in multiple myeloma (MM) and its underlying molecular basis. Materials and Methods: TRIM56 expression at the mRNA and protein level was measured by qRT PCR and western blot analysis. 3-(4,5-dimethylthiazol-2-yl)-2,5-diphenyltetrazolium bromide (MTT) and flow cytometry analysis was performed to investigate the effect of TRIM56 on MM cell proliferation and apoptosis. The concentrations of interferon (IFN)-β, interleukin (IL)-6, and tumor necrosis factor-α in MM cell culture supernatants were detected with respective commercial ELISA kits. Western blot was employedto determine the effect of TRIM56 on toll-like receptor 3 (TLR3)/toll-IL-1 receptor (TIR) domain-containing adaptor inducingIFN-β (TRIF) signaling pathway. Results: TRIM56 expression was prominently decreased in MM cells. Poly (dA:dT)-induced TRIM56 overexpression in U266 cells suppressed proliferation, induced apoptosis, and enhanced inflammatory cytokine production, while TRIM56 knockdown improvedgrowth, diminished apoptosis, and inhibited inflammatory cytokine secretion in RPMI8226 cells. Moreover, TRIM56 knockdownblocked TLR3 signaling pathway. Furthermore, poly (I:C), a TLR3 agonist, markedly abolished TRIM56 depletion-induced increase of proliferation, decrease of apoptosis, and reduction of inflammatory factor in MM cells. Conclusion: TRIM56 may act as a tumor suppressor in MM through activation of TLR3/TRIF signaling pathway, contributing to a better understanding of the molecular mechanism of TRIM56 involvement in MM pathogenesis and providing a promising therapystrategy for patients with MM.
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