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S. Y. Anaman,S. Ansah,M. G. Jo(조민구),J. Y. Suh(서진유),H. N. Han(한흥남),M. J. Kang(강민정),J. S. Lee(이종숙),S. T. Hong(홍성태),H. H. Cho(조훈휘) 한국소성가공학회 2021 한국소성가공학회 학술대회 논문집 Vol.2021 No.5
The electrochemical properties of a friction stir processed (FSPed) equiatomic CrMnFeCoNi high-entropy alloy (HEA) is investigated in an aerated 0.5 M Na<sub>2</sub>SO<sub>4</sub> electrolyte solution at room temperature. The microstructural analysis reveals a highly refined stir zone (SZ) with an average grain size that decreases from the top region of the SZ to the bottom region of the SZ (also known as shear processed zone; SPZ). However, the region below the SPZ, (i.e., below the plunge depth) experienced an increase in average grain size and dislocation densities compared to the other regions. There is no secondary phase observed in the FSPed region, however, the microstructural evolution in the FSPed region affects the electrochemical behavior of the HEA. Cr<sub>2</sub>O<sub>3</sub> passive layer is observed to form on the FSPed HEA leading to excellent corrosion properties from the polarization corrosion tests. Grain refinement in the SZ enhances the rapid formation of the passive layer, thus, leading to better corrosion properties in the front surface of the FSPed HEA. The localized corrosion behavior of the FSPed HEA is predicted to be caused by the micro-galvanic nature of the HEA, which leads to an increase in polarization at the anodic sites (pits). A numerical model is established using the corrosion parameters from the experiment to simulate the localized corrosion behavior on the surface of the FSPed HEA in a neutral environment. The predicted initial pitting potential and corresponding current density agree well with the experimental results. The model is also capable of tracking the dissolution of the pits over longer periods.
원저(原著) : 혈소판 농축액 교차시험에서 발견된 항-E+c+Dia의 multiple antibody 검출 1예
이종숙 ( J. S. Lee ),구미숙 ( M. S. Ku ),임연희 ( Y. H. Lim ),박주희 ( J. H. Park ),홍인표 ( I. P. Hong ) 대한임상병리사협회 1997 임상수혈검사학회 발표자료집 Vol.4 No.1
The Diego blood group system is controlled by two allelic genes, Di(a) and Di(b). Di(a) is known to be absent in pure Caucasians but present exclusively among Amerio-can Indians and Mongolians. The incidences of Di(a) antigen among Koreans are reported to be 6.14 to 14.5%. Recently the authors experienced a case with anti-Di(a) in a patient serum, which detected by routine bromelin cross-matching test selected with R1R1 blood for platelet concentrates. The case detected by irregular antibody identification test at an antiglobulin phase. Previously the patient was been detected anti-E and anti-c, had been transfused with R1R1 cells during the treatment for Aplastic Anemia, probably contributing to the presence of irregular antibody "anti ? Di(a)". After the identification of anti-E+c+Di(a) antibody the patient has been transfused with the selected compatible R1R1 Di(a-b+) blood, which could prevent the possible delayed hemolytic transfuion reaction.
이종숙 ( J S Lee ),박희정 ( H J Park ),구미숙 ( M S Ku ),홍인표 ( I P Hong ) 대한임상검사과학회 1991 대한임상검사과학회지(KJCLS) Vol.23 No.1
Autoantibody react on antigen existing on the surface of RBC, itself. Particularly, we considered warm autoantibody. The specificity of autoantibodies associated with warm autoimmune homolytic anemia is compl,~ x. so blood transfusion is very difficult. The authors experienced a case of autoimmune hemolytic anemia that caused by Rh-related antibody. No autoantibody specificity was clear out. But we could prove that the patient``s serum have Rh -related specificity with a difference of intencity of serum reaction. Special test proved major antibody are an ti-E & c. Blood transfusion was unavailable. We were . helped that selected compatible blood with crossmatching of R1 Rt cell in stead of many crossmatching. So we have reported warm autoantibody identification methods and signifirance of transfusion with brief review of literatures.
원저(原著) : ABO-HDN에서 검출된 Immune Anti-A(B)의 예
구미숙 ( M. S. Kim ),강대혁 ( D. H. Kang ),임연희 ( Y. H. Lim ),이종숙 ( J. S. Lee ),홍인표 ( I P. Hong ) 대한임상병리사협회 1996 임상수혈검사학회 발표자료집 Vol.3 No.1
Compatibility testing of ABO-HDN must be albumin Coombs cross matching, because of decting the immune Anti-A(B) from the mothers serum. IgG is the dominant form of Anti-A(B) of group O serum. The IgG form the placenta and can cause ABO-HDN. In ABO hemolytic disease of the newborn, when the DAT is only weakly positive or even negative, eluates from the infants red cell may give strong indirect antiglobulin reactions. HDN due to Anti-A(B) is observed only in infants who are enetically A1, At the time of birth the red sells of such infants may fail to react with Anti-A., although samples taken when the infant is a few months old do react. The case of this finding has never been demonstrated but blacking of antiqen sits by bound IgG Anti-A seems to be a possibility.