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RISS 인기검색어
- 검색키워드
- 저자 : Huili Zhu (검색결과 3 건)
- 무료
- 기관 내 무료
- 유료
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Use of GIS in the Numerical Emulation of the Fissure-Karster Water and the Optimum of Seepage Field
Huili, Gong,Wenji, Zhao,Yunqiang, Zhu 대한원격탐사학회 2001 International Symposium on Remote Sensing Vol.17 No.1
GTS represents the spatial features of the seepage field, while the numerical emulation models of the fissure-karst water is concerned with the flow of water and the flow of groundwater environment. In many cases, the numerical emulation models of the fissure-karst water assume uniform spatial properties or allow for small numbers of spatial subunits within which properties are uniform. GIS offers the potential to increase the degree of definition of spatial subunits, in number and in descriptive detail. To utilize these abilities, the conditions of hydrogeology are analyzed in the Zhuliu area, and the numerical emulation models of the fissure-karat water are built . The groundwater flow is emulated with the seepage model of heterogeneity anti-isotope. The partial differential equations are solved with finite difference scheme of irregular triangle net. Firstly, the inverse problem is solved for parameters calculating; then the positive problem is solved to calculate groundwater resources. By optimal control seepage field of the groundwater, the optimal exploitation quantity in overall research area cats be obtained.
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Structural Optimization of Compact Spherical Wind-Solar Hybrid Power System
Wei Huili,Pan Tianhong,Zhu Mingxing,Tao Jun 대한전기학회 2021 Journal of Electrical Engineering & Technology Vol.16 No.5
Conventional wind-solar hybrid power systems (WS-HPSs) have certain structural drawbacks owing to their large size and the difficulty in adjusting the tilt angle of the solar panels. To address these limitations, this study proposes a compact spherical wind-solar hybrid power system (CSWS-HPS). Furthermore, to investigate the aerodynamic performance of the designed CSWS-HPS, a computational fluid dynamics model of the wind rotor was established using the Reynolds-averaged Navier–Stokes equations, renormalization group k-ε turbulence model, and sliding mesh. Subsequently, the flow field distribution of velocity and pressure under different numbers of blades, blade installation angles, and tip-speed ratios (TSRs) were analyzed by performing a three-dimensional simulation of the CSWS-HPS. The monitored values of the moment coefficients were used to calculate the power coeffi cient value of the wind turbine to obtain the optimum structural parameters, which in turn provided the optimal values for the CSWS-HPS model. The simulation results revealed that the CSWS-HPS achieved considerable power generation efficiency in comparison with that of conventional hybrid systems. In addition, the CSWS-HPS is more compact in size and does not emit CO 2.
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Linyue Pan,Yuting Tan,Bin Wang,Wenjia Qiu,Yulei Yin,Haiyan Ge,Huili Zhu 대한암학회 2020 Cancer Research and Treatment Vol.52 No.3
Purpose Caspase recruitment domain containing protein 9 (CARD9) has been demonstrated to be a pro-tumor factor in various cancers. However, our previous study found a significant decrease of CARD9 in malignant pleural effusion compared with benign pleural effusion. So we investigated the role of CARD9 in non-small cell lung cancer (NSCLC) and its working mechanism. Materials and Methods Immunohistochemistry, western blot, and quantitative real-time polymerase chain reaction were used to detect the expression of CARD9 in specimens of NSCLC patients. The Cancer Genome Atlas (TCGA) database was also used to analyze the expression of CARD9 in NSCLC and its predicting value for prognosis. Immunofluorescence was used for CARD9 cellular location. Cell growth assay, clonal formation assay, wound healing assay, matrigel invasion assay, and flow cytometry were used to test cell proliferation, migration, invasion, apoptosis, and cycle progression of NSCLC cells with CARD9 knockdown or CARD9 overexpression. Co-immunoprecipitation was used to identify the interaction between CARD9 and B-cell lymphoma 10 (BCL10). SB203580 was used to inhibit p38 activation. Results CARD9 was decreased in NSCLC tissues compared with normal tissues; low CARD9 expression was associated with poor survival. CARD9 was expressed both in tumor cells and macrophages. Downregulation of CARD9 in NSCLC cells enhanced the abilities of proliferation, invasion, and migration via activated mitogen-activated protein kinases (MAPK)/p38 signaling, while overexpression of CARD9 presented antitumor effects. BCL10 was identified to interact with CARD9. Conclusion We demonstrate that CARD9 is an independent prognostic factor in NSCLC patients and inhibits proliferation, migration, and invasion by suppressing MAPK/p38 pathway in NSCLC cells.
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