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( Soo Jung Um ),( Su Mi Lee ),( Soo Keol Lee ),( Choon Hee Son ),( Mee Kyung Ko ),( Mee Sook Roh ),( Ki Nam Lee ),( Pil Jo Choi ) 대한결핵 및 호흡기학회 2011 Tuberculosis and Respiratory Diseases Vol.70 No.4
Background: It is well-known that cell-free nucleic acids rise in patients with many types of malignancies. Several recent experimental studies using cancer cell lines have shown that changes in cell-free RNA are predictive of the response to chemotherapy. The objective of this study was to determine whether quantification of free RNA can be used as a biomarker for clinical responses to chemotherapy in patients with lung cancer. Methods: Thirty-two patients with lung cancer (non-small cell lung cancer, n=24; small cell lung cancer, n=8) were divided into 2 groups according to their responses to chemotherapy (response group, n=19; non-response group, n=13). Blood samples were collected before and after two cycles of chemotherapy. Real-time quantitative RT-PCR was used for transcript quantification of the glyceraldehyde-3-phosphate dehydrogenase gene. Results: The pre chemotherapy values (Response group 41.36±1.72 vs. Non-response group 41.33±1.54, p=0.78) and post chemotherapy values (Response group 39.92±1.81 vs. Non-response group 40.41±1.47, p=0.40) for cell free RNA concentrations, expressed as Ct GAPDH (threshold cycle glyceraldehyde-3-phosphate dehydrogenase gene) levels, was not different between the two groups. There was no significant relationship between changes in the cell free RNA level clinical responses after chemotherapy (p=0.43). Conclusion: We did not find a correlation between quantification of serum cell free RNA levels and clinical responses to chemotherapy in patients with lung cancer. Further investigations are needed to determine whether the cell free RNA level is a useful predictor of responses to chemotherapy in patients with lung cancer.
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이수걸(Soo Keol Lee),조재웅(Jae Woong Cho),김선신(Sun Sin Kim),손지웅(Jee Woong Sohn),정기석(Ki Suck Jung),김익태(Ic Tae Kim),남동호(Dong Ho Nahm),박해심(Hae Sim Park) 대한천식알레르기학회 2000 천식 및 알레르기 Vol.20 No.4
Curry powder is widely used in various dishes. It is a mixture of 20-50 kinds of natural spices made from the leaves and seeds of plants. Although there have been some reports of allergy caused by inhalation of spices in western countries. While there are no reports of occupational allergy caused by spices in our country, We report a patient with rhinitis and asthma induced by exposure to spice dusts in a curry industry. A 32-year-old man developed rhinorrhea, sneezing and coughing three years prior to visiting our hospital. Since 10 years ago, he has been involved in grinding and mixing spices in a curry industry. Total peripheral eosinophil count was 400/mm and serum total IgE level was 163 IU/ml. Allergy skin-prick test showed positive responses to mugwort(3+), D. farinae(3+) and celery(3+), while serum specific IgE detected by RIA(DPC, LA, CA) shawed all negative results. Skin-prick test to four kinds of spice extracts- celery seed, fennel. cumin and coriander-showed strong positive responses. Bronchoprovocation test with celery seed extract(1: 10 v/v) showed an early asthmatic response. Specific IgE and IgG4 antibodies to celery seed and the other three spices were detectable by ELISA. IgE-ELISA inhibition test using each spice antigen showed significant inhibitions. In conclusion, IgE-mediated mechanism may be involved in the pathogenesis of curry powder-induced bronchoconstriction in an exposed worker. Further studies will be needed to investigate the role of specific IgG. in pathogenesis of bronchoconstriction in curry powder- induced asthma. (J Asthma Allergy Clin Immunol 20: 655-660, 2000)
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이수걸(Soo Keol Lee),서정희(Jung Hee Suh),김선신(Sun Sin Kim),남동호(Dong Ho Nahm),박해심(Hae Sim Park) 대한천식알레르기학회 2000 천식 및 알레르기 Vol.20 No.2
Inhalation of wood dust can cause immediate and/or late onset asthma, and the number of differeut kinds of wood responsible for such reactions is increasing. We report a patient with asthma induced by exposure to the dust of Japanese cedar wood (Cryptomeria japonica). A 30- year-old man developed rhinorrhea, sneezing and coughing, 6 years prior to admission. For the past 8 years, he had been involved in interior decoration of kitchen and sauna room using wood. Total peripheral eosinophil count was 600/mm3 and serum total IgE level was 505 IU/ml. Allergy skin prick test showed negative responses to 80 common inhalant and food allergens. Skin prick test to Japanese cedar wood dust extract showed negative response. Bronchoprovocation test with Japanese cedar wood dust extract (1:10 w/v) showed dual asthmatic response. Specific IgE and IgG4 antibodies to Japanese cedar wood dust extract were not detectable by ELISA. Serum neutrophil chemotactic assay using patients serum collected during bronchoprovocation test showed increased neutrophil chemotactic activity at 4 hr. In conclusion, non-IgE mediated mechanism may be involved in pathogenesis of Japanese cedar wood dust-induced asthma. Further studies will be needed to investigate the pathogenetic mechanism.
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A Transient Modeling Of A Fluorescent Lamp At Startup Time
Jung-Keol Ham,Soo Young Lee,Soo Hyun Baek,Ki Young Lee 전력전자학회 1995 ICPE(ISPE)논문집 Vol.1995 No.10
Fluorescent lamps are widely accepted to energy efficient commercial lighting applications. In designing a fluorescent lamp system, a ba11ast des ign heavi1y re1ies on the characterist ic of a fluorescent 1 amp under cons i derat i on. Espe-cially, at startup time, the transient characteristic of a fluorescent lamp puts much tighter specifications of a design.<br/> In this paper, based on the transient characteristic at the startup time, a transient behavioral model of a fluorescent lamp is presented wi th an equivalent circuit. The model is applicable to the wide range of f1uorescent 1 amps provided by different manufacturers.<br/> The experimental results are compared with the results provided by PSPICE simulation. The result shows the mode 1 i s effect ive i n practice. As a result, we could identify more accurate startup constraints to decide the design of either an electromechanical or an electronic ballast.
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