http://chineseinput.net/에서 pinyin(병음)방식으로 중국어를 변환할 수 있습니다.
변환된 중국어를 복사하여 사용하시면 됩니다.
- 中文 을 입력하시려면 zhongwen을 입력하시고 space를누르시면됩니다.
- 北京 을 입력하시려면 beijing을 입력하시고 space를 누르시면 됩니다.
RISS 인기검색어
- 검색키워드
- 저자 : Solá (검색결과 3 건)
- 무료
- 기관 내 무료
- 유료
-
Li, Yuelong,Carretero-Palacios, Sol,Yoo, Kicheon,Kim, Jong Hak,Jimé,nez-Solano, Alberto,Lee, Chul-Ho,Mí,guez, Herná,n,Ko, Min Jae The Royal Society of Chemistry 2016 Energy & environmental science Vol.9 No.6
<P>We demonstrate that a combined optimization approach based on the sequential alternation of theoretical analysis and experimental realization gives rise to plastic supported dye solar cells for which both light harvesting efficiency and electron collection are maximized. Rationalized configurations with optimized light trapping and charge extraction are realized to achieve photoanodes on plastic prepared at low temperature, showing a power conversion efficiency of 8.55% and a short circuit photocurrent of 16.11 mA cm(-2), unprecedented for plastic based dye solar cell devices. Furthermore, the corresponding fully flexible designs present stable mechanical properties after several bending cycles, displaying 7.79% power conversion efficiency, an average broadband internal quantum efficiency above 90%, and a short circuit photocurrent of 15.94 mA cm(-2), which is the largest reported value for bendable cells of this sort to date.</P>
-
-
Sebastian, Jose,Ryu, Kook Hui,Zhou, Jing,Tarkowská,, Danuš,e,Tarkowski, Petr,Cho, Young-Hee,Yoo, Sang-Dong,Kim, Eun-Sol,Lee, Ji-Young Public Library of Science 2015 PLoS genetics Vol.11 No.3
<▼1><P>Plant growth depends on stem cell niches in meristems. In the root apical meristem, the quiescent center (QC) cells form a niche together with the surrounding stem cells. Stem cells produce daughter cells that are displaced into a transit-amplifying (TA) domain of the root meristem. TA cells divide several times to provide cells for growth. SHORTROOT (SHR) and SCARECROW (SCR) are key regulators of the stem cell niche. Cytokinin controls TA cell activities in a dose-dependent manner. Although the regulatory programs in each compartment of the root meristem have been identified, it is still unclear how they coordinate one another. Here, we investigate how PHABULOSA (PHB), under the posttranscriptional control of SHR and SCR, regulates TA cell activities. The root meristem and growth defects in <I>shr</I> or <I>scr</I> mutants were significantly recovered in the <I>shr phb</I> or <I>scr phb</I> double mutant, respectively. This rescue in root growth occurs in the absence of a QC. Conversely, when the modified <I>PHB</I>, which is highly resistant to microRNA, was expressed throughout the stele of the wild-type root meristem, root growth became very similar to that observed in the <I>shr</I>; however, the identity of the QC was unaffected. Interestingly, a moderate increase in <I>PHB</I> resulted in a root meristem phenotype similar to that observed following the application of high levels of cytokinin. Our protoplast assay and transgenic approach using <I>ARR10</I> suggest that the depletion of TA cells by high PHB in the stele occurs via the repression of B-ARR activities. This regulatory mechanism seems to help to maintain the cytokinin homeostasis in the meristem. Taken together, our study suggests that PHB can dynamically regulate TA cell activities in a QC-independent manner, and that the SHR-PHB pathway enables a robust root growth system by coordinating the stem cell niche and TA domain.</P></▼1><▼2><P><B>Author Summary</B></P><P>Plant roots are programmed to grow continuously into the soil, searching for nutrients and water. The iterative process of cell division, elongation, and differentiation contributes to root growth. The quiescent center (QC) is known to maintain the root meristem, and thus ensure root growth. In this study, we report a novel aspect of root growth regulation controlled independently of the QC by PHABULOSA (PHB). In <I>shr</I> mutant plants, PHB, which in the meristem is actively restricted to the central region of the stele by SHORTROOT (SHR) via <I>miR165/6</I>, suppresses root meristem activity leading to root growth arrest. A high concentration of PHB in the stele does this by modulating B-ARR activity through a QC-independent pathway. Accordingly, we observed a significant recovery of root meristem activity and growth in the <I>shr phb</I> double mutant, while the QC remained absent. However, the presence of QC may be required to sustain continuous root growth. On the basis of our results, we propose that SHR maintains root growth via two separate pathways: by modulating PHB levels in the root stele, and by maintaining the QC identity.</P></▼2>
연관 검색어 추천
이 검색어로 많이 본 자료
활용도 높은 자료
