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Air–Liquid Interfacial Self-Assembly of Non-Amphiphilic Poly(3-hexylthiophene) Homopolymers
Oh, Saejin,Yang, Myungjae,Bouffard, Jean,Hong, Seunghun,Park, So-Jung American Chemical Society 2017 ACS APPLIED MATERIALS & INTERFACES Vol.9 No.14
<P>Here, we demonstrate that the self-assembly of poly(3-hexylthiophene) (P3HT) at the air-water interface can lead to free-standing films of densely packed P3HT nanowires. Interfacial self-assembly on various liquid subphases, such as water, diethylene glycol, and glycerol, indicates that the-viscosity of the subpliase is an important factor for the formation of well-ordered nanostructures. The thin-film morphology is also sensitive to the concentration of P3HT, its molecular weight (MW), and the presence of oxidative defects. The densely packed nanowire films can be easily transferred to solid substrates for device applications. The ultrathin films of P3HT prepared by the interfacial assembly showed significantly higher hole mobility (similar to 3.6 X 10(-2) cm(2)/V s) in a field-effect transistor than comparably thin spin-cast films. This work demonstrates that the air liquid interfacial assembly is not limited to amphiphilic polymers and can, under optimized conditions, be applied to fabricate ultrathin films of widely used conjugated polymers with controlled morphologies.</P>
Diethylnitrosamine에 의하여 유발된 마우스 간 종양의 CYP1A2 메틸화와 발현
진보환(Bohwan Jin),오새진(Saejin Oh),류덕영(Doug-Young Ryu) 한국환경성돌연변이발암원학회 2006 한국환경성돌연변이·발암원학회지 Vol.26 No.3
Cytochrome P450 1A2 (CYP1A2) is a xenobiotic metabolizing enzyme that is tissue-specifically expressed in the mammalian liver. In this study, the extent of CYP1A2 promoter methylation was analyzed to determine its potential role in the regulation of CYP1A2 in diethylnitrosamine (DEN)-induced mouse liver tumors. CYP1A2 mRNA was under-expressed about three fold in DEN-induced liver tumors compared to agematched control livers. The CYP1A2 promoter was hypermethylated in DEN-induced liver tumors compared to controls, especially in a promoter domain close to the coding region. These results suggest that promoter methylation is involved in the regulation of CYP1A2 in mouse liver tumors.