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High-performance discrete amplifier based on a second-order fiber Raman oscillator
Faralli, S.,Kim, Pilhan,Cantini, C.,Bolognini, G.,Sacchi, G.,Park, Namkyoo,Di Pasquale, F. IEEE 2005 IEEE photonics technology letters Vol.17 No.11
We propose a second-order counterpumped Raman ring fiber oscillator for high gain and low noise discrete Raman amplification with enhanced robustness to wavelength-division-multiplexing channels add-drop operations. Bit-error-rate measurement at 10 Gb/s shows negligible penalties up to 20-dB net gain.
The Situation Lens: A Metaphor for Personal Task Management on Mobile Devices
Celentano, Augusto,Faralli, Stefano,Pittarello, Fabio Korean Institute of Information Scientists and Eng 2009 Journal of Computing Science and Engineering Vol.3 No.4
In this paper we discuss personal data management with mobile devices, an activity requiring the composition of services offered by standard suites of applications. We propose a data model and an interface model that allows users to define activities, tasks and services, to navigate among them according to the evolution of the personal situation as perceived and interpreted by the users themselves. The interface model acts as a lens exploring the situation, zooming into the details, covering different areas of the personal data, supporting the user in the role of a composer of personal services.
The Situation Lens : A Metaphor for Personal Task Management on Mobile Devices
Augusto Celentano,Stefano Faralli,Fabio Pittarello 한국정보과학회 2009 Journal of Computing Science and Engineering Vol.3 No.4
In this paper we discuss personal data management with mobile devices, an activity requiring the composition of services offered by standard suites of applications. We propose a data model and an interface model that allows users to define activities, tasks and services, to navigate among them according to the evolution of the personal situation as perceived and interpreted by the users themselves. The interface model acts as a lens exploring the situation, zooming into the details, covering different areas of the personal data, supporting the user in the role of a composer of personal services.
A KAP1 phosphorylation switch controls MyoD function during skeletal muscle differentiation
Singh, Kulwant,Cassano, Marco,Planet, Evarist,Sebastian, Soji,Jang, Suk Min,Sohi, Gurjeev,Faralli, Hervé,Choi, Jinmi,Youn, Hong-Duk,Dilworth, F. Jeffrey,Trono, Didier Cold Spring Harbor Laboratory Press 2015 Genes & development Vol.29 No.5
<P>The transcriptional activator MyoD serves as a master controller of myogenesis. Singh et al. identify KAP1/TRIM28 as a key regulator of MyoD function. In myoblasts, KAP1 is present with MyoD and Mef2 at many muscle genes, where it acts as a scaffold to recruit not only coactivators such as p300 and LSD1 but also corepressors such as G9a and HDAC1, with promoter silencing as the net outcome. Upon differentiation, MSK1-mediated phosphorylation of KAP1 releases the corepressors from the scaffold, unleashing transcriptional activation by MyoD/Mef2 and their positive cofactors.</P><P>The transcriptional activator MyoD serves as a master controller of myogenesis. Often in partnership with Mef2 (myocyte enhancer factor 2), MyoD binds to the promoters of hundreds of muscle genes in proliferating myoblasts yet activates these targets only upon receiving cues that launch differentiation. What regulates this off/on switch of MyoD function has been incompletely understood, although it is known to reflect the action of chromatin modifiers. Here, we identify KAP1 (KRAB [Kréééüppel-like associated box]-associated protein 1)/TRIM28 (tripartite motif protein 28) as a key regulator of MyoD function. In myoblasts, KAP1 is present with MyoD and Mef2 at many muscle genes, where it acts as a scaffold to recruit not only coactivators such as p300 and LSD1 but also corepressors such as G9a and HDAC1 (histone deacetylase 1), with promoter silencing as the net outcome. Upon differentiation, MSK1-mediated phosphorylation of KAP1 releases the corepressors from the scaffold, unleashing transcriptional activation by MyoD/Mef2 and their positive cofactors. Thus, our results reveal KAP1 as a previously unappreciated interpreter of cell signaling, which modulates the ability of MyoD to drive myogenesis.</P>