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Zahra, Zahra,Waseem, Naima,Zahra, Rubab,Lee, Hwanhui,Badshah, Mohsin Ali,Mehmood, Arshad,Choi, Hyung-Kyoon,Arshad, Muhammad American Chemical Society 2017 Journal of agricultural and food chemistry Vol.65 No.28
<P>Plants have the natural ability to withstand stress conditions through metabolic adjustments. The present study aimed at investigating the effects of titanium dioxide nanoparticles (TiO2 NPs) application (0, 25, 50, 150, 250, 500, and 750 mg kg(-1)) in phosphorus-deficient soil in terms of growth responses, P contents, and metabolic alterations in rice. TiO2 NPs application increased shoot length up to 14.5%. Phosphorus contents in rice roots, shoots, and grains were increased by 2.6-, 2.4-, and 1.3-fold, respectively, at 750 mg kg(-1) of TiO2 NPs. Gas chromatography-mass spectrometry (GC-MS)-based metabolomics revealed increased levels of amino acids, palmitic acid, and glycerol content in grains resulting from plants grown in 750 mg kg(-1) TiO2 NPs-treated soil. Furthermore, no translocation of TiO2 NPs from the treated soil to rice grains was detected by inductively coupled plasma-optical emission spectrometry (ICP-OES), which suggests no risk of TiO2 NPs intake via grain consumption. The observed data indicates the strong relationship among NPs application, P contents, and metabolic alterations.</P>
Zahra, Zahra,Kim, Seok-Young,Kim, Hye-Youn,Lee, Hwanhui,Lee, Heayyean,Jeon, Jun-Yeong,Kim, Dong-Min,Kim, Dong-Myung,Hong, Seong-Joo,Cho, Byung-Kwan,Lee, Hookeun,Lee, Choul-Gyun,Arshad, Muhammad,Choi, American Chemical Society 2018 Journal of agricultural and food chemistry Vol.66 No.32
<P>This study aimed to improve the production of phycobiliproteins using TiO<SUB>2</SUB> nanoparticles (NPs) in <I>Synechocystis</I> sp. PCC 6803. The growth characteristics of <I>Synechocystis</I> cells were not affected by TiO<SUB>2</SUB> NPs treatment, but this treatment increased the chlorophyll content significantly by 62.2% (14.6 mg/L) compared to that of control (9.0 mg/L) on day 16. Phycocyanin production was increased by 33.8% (29.3 g/L) compared to that of control (21.9 g/L) on day 8. Allophycocyanin production was increased by 55.0% (6.2 g/L) compared to that of control (4.0 g/L) on day 8, and by 22.4% (16.4 g/L) compared to that of control (13.4 g/L) on day 16. Direct infusion mass spectrometry revealed that TiO<SUB>2</SUB> NPs treatment significantly increased the levels of major thylakoid membranes of monogalactosyldiacylglycerols (18:2/18:3, 18:2/18:2, 18:1/18:2), phosphatidylglycerol (16:0/16:1), and sulfoquinovosyldiacylglycerols (16:0/16:1, 16:0:18:4) on day 8. These findings indicate that TiO<SUB>2</SUB> NPs have potential for commercial applications in <I>Synechocystis</I> species or other microalgal strains.</P> [FIG OMISSION]</BR>
Zahra Azimzadeh(Zahra Azimzadeh ),Samareh Omidvari(Samareh Omidvari ),Somayeh Niknazar(Somayeh Niknazar ),Saeed Vafaei-Nezhad(Saeed Vafaei-Nezhad ),Navid Ahmady Roozbahany(Navid Ahmady Roozbahany ),Mo 대한해부학회 2024 Anatomy & Cell Biology Vol.57 No.1
Methamphetamine (METH) can potentially disrupt neurotransmitters activities in the central nervous system (CNS) and cause neurotoxicity through various pathways. These pathways include increased production of reactive nitrogen and oxygen species, hypothermia, and induction of mitochondrial apoptosis. In this study, we investigated the long-term effects of METH addiction on the structural changes in the amygdala of postmortem human brains and the involvement of the brain- cAMP response element-binding protein/brain-derived neurotrophic factor (CREB/BDNF) and Akt-1/GSK3 signaling pathways. We examined ten male postmortem brains, comparing control subjects with chronic METH users, using immunohistochemistry, real-time polymerase chain reaction (to measure levels of CREB, BDNF, Akt-1, GSK3, and tumor necrosis factor-α [TNF-α]), Tunnel assay, stereology, and assays for reactive oxygen species (ROS), glutathione disulfide (GSSG), and glutathione peroxidase (GPX). The findings revealed that METH significantly reduced the expression of BDNF, CREB, Akt-1, and GPX while increasing the levels of GSSG, ROS, RIPK3, GSK3, and TNF-α. Furthermore, METH-induced inflammation and neurodegeneration in the amygdala, with ROS production mediated by the CREB/BDNF and Akt-1/GSK3 signaling pathways.